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Titlebook: Clostridium difficile; Methods and Protocol Peter Mullany,Adam P. Roberts Book 2010 Springer Science+Business Media, LLC 2010 DNA.Microarra

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https://doi.org/10.1007/978-3-642-83960-3cell wall and its components also have to mediate interactions with the host cells and their products. In this chapter we discuss the various methods used for dissecting the cell surface and the biochemical and immunological procedures that are commonly used to analyse the properties of the proteins
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https://doi.org/10.1007/978-3-642-83960-3itis, particularly in hospitalized, elderly patients. Studies to date suggest that . toxins A and B play a major role in the observed colonic inflammation and associated disease pathogenesis; however, the role of other potential bacterial factors at present remains unknown. Early effects of . on hos
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Was verursacht Schizophrenien?,ological processes they undertake. This has led to increasing efforts directed at deriving greater information on their basic biology, most notably through genome sequence. Accordingly, annotated sequences of all of the most important species are now available. However, full exploitation of the data
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,Das heutige Wissen um „Schizophrenien“,ulent” strains associated with increased rates of mortality and severity of disease in humans has highlighted the need to study this organism at the molecular level. These studies will increase our knowledge of the mechanisms by which . causes disease and facilitate the rational design of new and im
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https://doi.org/10.1007/978-3-662-09811-0at is to use the conjugative transposon Tn.. Tn. enters the genome of most strains of . with no obvious target site preference. In order to use the genome strain . 630 for transposon mutagenesis a erythromycin-sensitive derivative . 630Δ. was constructed and the Tn. derivative, Tn.ΔE, was shown to e
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