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Titlebook: Clinical Diagnosis of Atherosclerosis; Quantitative Methods M. Gene Bond (Associate Professor of Comparative M Textbook 1983 Springer-Verla

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发表于 2025-3-21 18:19:25 | 显示全部楼层 |阅读模式
书目名称Clinical Diagnosis of Atherosclerosis
副标题Quantitative Methods
编辑M. Gene Bond (Associate Professor of Comparative M
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图书封面Titlebook: Clinical Diagnosis of Atherosclerosis; Quantitative Methods M. Gene Bond (Associate Professor of Comparative M Textbook 1983 Springer-Verla
描述This volume is the product of a February 1982 conference, cosponsored by the American Heart Association, the National Institutes of Health, and the Bowman Gray School of Medicine, which examined techniques for delineating quantitatively the natural history of atherosclerosis. Against the background of current pathologic and clinical knowledge of atherosclerosis, invasive and noninvasive evaluative methods now in use and under development are surveyed in depth. Correlative clinicopathologic studies of atherosclerosis pose special questions with respect to both luminal and plaque characteristics that are addressed in this volume. An old observa­ tion, based on the examination of arterial casts, suggested that the so-called nodose lesion of atherosclerosis may be at first flattened into the wall of a weakened, dilated artery, rather than raised into the lumen. This is now fully confirmed in vivo by ultrasonic and other imaging techniques. The morbid anatomist is challenged anew to describe lesions as they are likely to occur in vivo. To achieve closer correlation with natural conditions, perfu­ sion fixation of arteries under arterial pressure is becoming more widely used and has alre
出版日期Textbook 1983
关键词anatomy; angiography; arterial pressure; atherosclerosis; heart; ultrasound
版次1
doihttps://doi.org/10.1007/978-1-4684-6277-7
isbn_softcover978-1-4684-6279-1
isbn_ebook978-1-4684-6277-7
copyrightSpringer-Verlag New York Inc. 1983
The information of publication is updating

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发表于 2025-3-21 23:30:29 | 显示全部楼层
https://doi.org/10.1057/9781403907172toms that simulate arteries in the millimeter range (1). While these noninvasive methods appear very likely to be used for quantitative lesion assessment in the near future, relatively little quantitative experience has been gained to date. Conversely, computer methods to quantify atherosclerosis fr
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https://doi.org/10.1057/9780230372931that lesion has increased to approximately 20% of the total mass. However, in carefully dissected, large, raised lesions described as atherosclerotic plaques, the percent dry weight is often greater than 50% (2). Since the density of the lipids is some 30–40% less than the density of the other const
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https://doi.org/10.1007/978-1-349-05265-3ection. Severe, uncompensated obstructions, on the other hand, compromise the viability of the tissues even at rest or in the absence of extraneous trauma--leading to ischemic rest pain, nonhealing ulcers, tissue destruction by unchecked infection, and eventual gangrene. Although lesions capable of
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https://doi.org/10.1007/978-3-319-70341-1 components during lesion initiation, progression, and regression. An appreciation of the biochemical and subcellular changes and spatial relations of lesion components at various points in time is essential to understanding the pathogenesis of atherosclerosis. The importance of these relationships
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Quantitating Atherosclerosis self-evident that controversies regarding the relative accuracy, sensitivity and specificity of each of the detection methods can ultimately be resolved only by quantitative comparisons of the images with the actual, corresponding lesions. Secondly, we have entered an era in which we wish to follow
发表于 2025-3-23 09:00:45 | 显示全部楼层
Atherosclerosis Quantitation by Computer Image Analysistoms that simulate arteries in the millimeter range (1). While these noninvasive methods appear very likely to be used for quantitative lesion assessment in the near future, relatively little quantitative experience has been gained to date. Conversely, computer methods to quantify atherosclerosis fr
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