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Titlebook: Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity; Santiago Grisolía,Vicente Felipo,María-Dolores Miñ Book 1990 The Editor(s) (if a

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https://doi.org/10.1007/978-1-4614-5419-9s or carrying out long-term evaluations to determine its protective effect in the appearance of hepatic encephalopathy, perhaps even including its evaluation in the treatment of established encephalopathy.
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Effect of L-Carnitine Upon Ammonia Tolerance Test in Cirrhotic Patientss or carrying out long-term evaluations to determine its protective effect in the appearance of hepatic encephalopathy, perhaps even including its evaluation in the treatment of established encephalopathy.
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Cerebral Function in Hepatic Encephalopathyas a metabolic etiology. Even in the absence of serious symptoms brain function is compromised and is more sensitive to a variety of metabolic disturbances which in normal individuals would cause no serious alterations in cerebral function (1,2).
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Ammonia Metabolism in Normal and Portacaval-Shunted Ratss ammonia is neurotoxic and its concentration must be kept low. Ammonia generated in most extrahepatic tissues is detoxified by incorporation into glutamine (amide). This glutamine may be used in a number of biosynthetic reactions (e.g. in pyrimidine synthesis). Alternatively, as a means of maintain
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Nitrogen Metabolism in Normal and Cirrhotic Liverwith metabolic interactions between different hepatocyte populations. This provided a new insight into the role of the liver in maintaining ammonia and bicarbonate homeostasis. A specialized cell population at the hepatic venous outflow of the liver acinus acts as a scavenger for ammonia and probabl
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