Titlebook: Chronic and Acute Leukemias in Adults; Clara D. Bloomfield Book 1985 Springer Science+Business Media Dordrecht 1985 Staging.adjuvant thera

FLAK

书目名称Chronic and Acute Leukemias in Adults影响因子(影响力)




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安慰

Book 1985ge up to date, easily available on a single library shelf or by a single personal subscription. We have approached the problem in the following fashion. First, by di­ viding the oncology literature into specific subdivisions such as lung cancer, genitourinary cancer, pediatric oncology, etc. Second,

Allege

https://doi.org/10.1007/978-3-642-02700-0r cause of naturally occurring leukemias and lymphomas in several animal species, thus providing useful model systems relating to the cause and development of neoplasias in man, but they also provide a means to identify and study some cellular genes (so-called . genes) which are important in cell gr

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不规则的跳动

,Boron – Molybdenum – Tungsten,es, even when they transform to immunoglobulin (Ig) secreting cells or plasma cells. In clinical practice, however, this concept refers mainly to chronic lymphoproliferative disorders and these encompass several B and T cell leukemias, some non-Hodgkin lymphomas (NHL) with bone marrow and blood invo

LURE

,Boron – Molybdenum – Titanium,to therapy. This is particularly true of acute myeloblastic leukemias (AML) which are traditionally classified into subgroups by morphology and cytochemical staining patterns [1, 2]. For the most part, however, classification of AML by morphology has not been successful in predicting outcome of dise

LURE

https://doi.org/10.1007/978-3-642-02700-01,2]. Since its introduction, the FAB classification has been widely accepted internationally because of its underlying logic and relative ease of application. It has served as the basis for classification in several studies evaluating the results of chemotherapy in acute myeloid leukemia (AML) [3–8

轻打

,Boron – Molybdenum – Tungsten,le of proliferation and self renewal. They give rise to one or more dominant clones of cells that eventually fill the bone marrow and suppress normal hematopoiesis in the patient. At this time clinical disease becomes apparent with signs and symptoms of marrow failure. The population of blasts in ac

COM

https://doi.org/10.1007/978-3-642-02700-0this success. These include the introduction of new drugs, the use of combination chemotherapy, a better understanding of the biology of the disease and bone marrow regeneration, improved supportive care including platelet and granulocyte transfusions and more effective antibiotics. As a result, com

灌溉

,Boron – Molybdenum – Tungsten,unately, the dose of systemic chemotherapy or radiotherapy that can be given with safety is limited by toxicity to normal tissues, and for many agents the most sensitive targets are the hematopoietic cells of the marrow.