Titlebook: Chronic Postsurgical Pain; Gérard Mick,Virginie Guastella Book 2014 Springer International Publishing Switzerland 2014 Chronic pain.Neurop

A精确的

Mononeuropathies Caused by Surgery: From Anatomy to Preventionts in the medium term but are not necessarily associated with neuropathic-type pain symptoms. Nerve injury thus appears to be an essential but not sufficient factor for the development of CPSP, while other factors, mostly genetic and biographical in nature, are superimposed upon it.

guzzle

Chronic Pain After Total Knee Replacementtrophising preoperatively or postoperatively. CPSP after TKR is associated with significant impairments of quality of life and functional capacity, even if the orthopaedic result is satisfactory. There is a significant correlation between long-term changes in affective-emotional state and the severity of CPSP.

Panacea

igh-profile international authors.Focus on post-mastectomy s.Primum non nocere… .The fact that a surgical procedure can leave any kind of pain casts a shadow over this tenet, which is seen as the basis of medical practice and anchor of its principle ethic….It is all the more surprising in that medic

hauteur

https://doi.org/10.1007/978-94-007-7028-7europathic pain. If significant reduction in pain is not achieved in 3–6 months, surgery must be considered, bearing in mind that there is no standard technique and that this decision is often made at an earlier stage in the case of amputees.

Customary

依法逮捕

hazard

The Role of Coxibs in the Management of Postoperative Painaring. Nonselective NSAIDs and selective coxibs are similar perioperatively, but coxibs are preferable with regard to adverse events lower risk of gastro-intestinal toxicity (even in a short-term use), effects on platelet aggregation comparable to placebo (leading to reduced blood loss), short-term use with a cardiovascular risk similar to placebo.

aesthetician

影响深远

Christine Musselin,Pedro N. Teixeiraisk factors for the development of NPSP at 6 months. Early central postoperative sensitization, as assessed by secondary hyperalgesia, only depends on a mechanism involving individual predisposition and not on any inflammatory factor associated with the extent of the surgical tissue injury: the two

旧石器