Titlebook: Chirurgisches Forum 2003 für experimentelle und klinische Forschung; 120. Kongress der De M. D. Menger (Direktor der Abteilung für Klinisch

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San Juana Aguas-Marmolejo,Oscar Castillon of tumour relevant proteins. We used a proteomic-based approach to search for tumour specific target molecules in pancreatic carcinoma tissue and present data on some promising candidates with respect to their tumour biological value. The expression of tumour-associated antigens can provoke an aut

backdrop

Bacterial Effectors: Learning on the Fly,se plasminogen activator receptor (uPAR). Hypoxia-Inducible-Factor-1 (HIF-1) is found to be activated during progression of human pancreatic cancer, a tumor which grows in a microenvironment of low oxygen. However, it is not know whether a relationship between HIF-1 and uPAR exists. Transcriptional

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思想

Hon Keung Tony Ng,Daniel F. Heitjan basis of PDAC, further studies are needed to find new molecular markers for diagnostic and therapeutic purposes. We have studied the mRNA-expression profile of microdissected PDAC, of microdissected normal pancreatic duct cells, of one primary normal pancreatic cell line, and of 5 established pancr

Meager

Omer Ozturk,Jennifer Brown,Olena Kravchukenograft tumor model for human colon carcinoma in athymic rats (HT-29a, HT-29c). We searched for differences of the protein levels by analyses of these cell lines by a parallel western-blot screening method (POWERBLOT). According to quantitative and qualitative differences, levels of confidence were

妨碍议事

Bivin Philip Sadler,S. Lynne Stokeses indicate a worse prognosis versus lymph node negative tumors. Accordingly, there is considerable clinical interest in understanding the genetic mechanisms underlying metastasis formation. To assess genomic imbalances, we used comparative genomic hybridization (CGH) in fifty colorectal cancers wit

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Yana Mazwin Mohmad Hassim,Rozaida Ghazali like e.g. TNM and UICC as shown for other tumor entities. The aim of this study was to identify prognostic signatures for patients with colorectal cancer of all UICC subgroups, especially for patients with versus without lymph node metastasis (N0 vs. N +), and for patients with versus without dista