Titlebook: Cerebral Sinus Thrombosis; Experimental and Cli Karl Einhäupl,Oliver Kempski,Alexander Baethmann Book 1990 Springer Science+Business Media

原谅

咯咯笑

A New Experimental Model of Sinus Vein Thrombosismbogenic material [1–3]. Ligation and injection of lard-oil or cyanoacrylate into the superior sagittal sinus (SSS) produced obstruction of the SSS, or of the superior cerebral veins (SCV) yet without evidence of thrombosis [4–7]. Materials such as scandium chloride, sodium morrhuate, or thrombin in

终点

FLORA

Brain Function, Pathophysiology and Heparin Therapy in Experimental Cerebral Sinus Vein Thrombosiserved only at autopsy raising the suspicion that SVT often is a lethal disorder. Recent experience indicates, however, that SVT has a wide clinical spectrum reaching from discrete neurological deficits to most severe courses with coma [1]. Contrary to findings for arterial stroke, even severe neurol

残忍

Physiology and Pathophysiology of the Cerebro-Venous Circulationernoy) [1]. Most of the cortical venous blood reaches the superficial pial veins, which have a rich network of short and long veno-venous connections [2]. The latter are draining into bridging veins, which enter the durai sinus. A number of large pial collateral veins, the Rolandic, Sylvian, Trolard

怒目而视

Cerebral Blood Flow in Patients with Sinus Vein Thrombosis into differential diagnostic considerations which then will lead to appropriate studies, eventually allowing the correct diagnosis to be made [4]. Apart from diagnostic difficulties, there is still a considerable lack of evidence to explain the underlying pathophysiologic mechanisms of cerebral SVT

大雨

Vascular Smooth Muscle Reactivity in Hypoxianal blood flow increases significantly if the arterial oxygen falls below 60 mmHg. In the presence of hypoxia severe enough to cause anoxia in single cells, characteristic metabolic changes develop in cerebral cortex. Therefore, adjustment of the vascular system under these conditions is usually att

壕沟

Mechanisms of Ischemic Brain Damaged veins. In some of these territories the supply of blood may fall below a critical threshold, resulting in an ischemic or hypoxemic lesion. Experimental SVT is indeed characterized by a significant rise of tissue electrical impedance [1] as an indication of ischemic cell swelling. Therefore, treatm

冲击力

URN

Eicosanoids, Peptides and Amines and the Cerebral Blood Vesselsts tight relationships between blood flow, oxygen and glucose metabolism and neuronal activity. Of primary importance in this regard are ionic species and metabolic products such as potassium, calcium, carbon dioxide and lactate which in spite of careful regulation might also be subjected to rapid a