Titlebook: Cellular Peptidases in Immune Functions and Diseases 2; Jürgen Langner,Siegfried Ansorge Book 2002 The Editor(s) (if applicable) and The A

摇摆

N-Terminal HIV-1 Tat Nonapeptides as Inhibitors of Dipeptidyl Peptidase IV. Conformational Characterffects of HIV-1 Tat, the Ile. and Leu. analogues showed strongly reduced inhibitory activity. Interestingly, replacement of Asp. with Gly or Lys led to compounds with considerably enhanced inhibition. Therefore, we have applied .H NMR spectroscopy and restrained molecular dynamics calculations to el

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偶像

Dipeptidyl Peptidase IV in Inflammatory CNS Disease autoimmune response is involved in the disease process. One of the primary goals in the in the development of immunotherapies for autoimmune diseases has been to achieve inactivation of disease-inducing lymphocytes either by direct inhibition or suppression through regulatory cells and/or cytokines

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Dipeptidyl Peptidase IV (CD26): Role in T Cell Activation and Autoimmune Disease show by flow cytometry and by enzymatic DP IV assay that myelin basic protein (MBP)-specific, CD4. T cell clones (TCC) derived from patients with multiple sclerosis (MS) express high levels of DP IV/CD26. The enzymatic activity of resting TCC was found to be three to fourfold higher than on resting

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DNA Synthesis in Cultured Human Keratinocytes and Hacat Kerationcytes is Reduced by Specific Inhibiten shown that this enzyme is involved in the regulation of DNA synthesis and in production of various cytokines in lymphocytes. The aim of the present work was to investigate the expression of DP IV/CD26 on human keratinocytes and to answer the question, whether the proliferation (DNA synthesis) of

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Cellular Peptidases in Immune Functions and Diseases 2978-0-306-46826-1Series ISSN 0065-2598 Series E-ISSN 2214-8019

Ordnance

0065-2598 Overview: 978-1-4757-8649-1978-0-306-46826-1Series ISSN 0065-2598 Series E-ISSN 2214-8019

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