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Titlebook: Cell Growth and Oncogenesis; Peter Bannasch (Head Division of Cell Pathology),D Book 1998 Springer Basel AG 1998 Activation.DNA.Viruses.ca

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书目名称Cell Growth and Oncogenesis
编辑Peter Bannasch (Head Division of Cell Pathology),D
视频videohttp://file.papertrans.cn/223/222827/222827.mp4
丛书名称Molecular and Cell Biology Updates
图书封面Titlebook: Cell Growth and Oncogenesis;  Peter Bannasch (Head Division of Cell Pathology),D Book 1998 Springer Basel AG 1998 Activation.DNA.Viruses.ca
描述Rapid progress has been made in our understanding of the molecular mechanisms of cell growth and oncogenesis during the past decade. This book comprises recent results on the regulation of cell growth in normal and neoplastic tissues by growth factors including hormones, and by the activation and inactivation of oncogenes and tumor suppressor genes, respectively. Special attention has been given to the presentation of the frequently neglected close correlation between changes in signal transduction and metabolism pathways during oncogenesis.
出版日期Book 1998
关键词Activation; DNA; Viruses; cancer; carcinogenesis; cell; cell division; glycoprotein; growth; hormones; metabol
版次1
doihttps://doi.org/10.1007/978-3-0348-8950-6
isbn_softcover978-3-0348-9841-6
isbn_ebook978-3-0348-8950-6
copyrightSpringer Basel AG 1998
The information of publication is updating

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The mitochondrial ATP synthase in normal and neoplastic cell growthply which can cover 50% or more of their energy requirement (Nakashima et al., 1984). This phenotype of cancer cells raises a number of questions. Which are the characteristics and the causes of the changes in the energy metabolism? Do they provide cancer cells with an advantage for their growth? If
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Hepatic regeneration: New concepts on cell proliferationesized role of the immune system on controlling it..In the present study we demonstrated in rats in the remaining liver after 70% partial hepatectomy (HP) a significant loss of cytotoxic ability by liver-derived mononuclear leukocytes which begins immediately after PH and continues for the next 14 d
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E7 protein of human papillomaviruses and its interaction with cellular pathwaysces, can be divided into three different regions: (a) a non-coding region (about 1000 bp), which contains several . and . elements that control viral replication and transcription; (b) the region that encodes the six early genes, El, E2, E4, E5, E6, E7; and (c) the region that encodes the two late g
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Phosphorylation of the p53 tumour suppressor protein by stress- and DNA damage-activated protein kinprotein which encompasses a number of activities, including a potent transactivation function. Activation of p53 is initiated by a range of stresses including DNA damage and leads to cellular growth arrest or programmed cell death. We have shown that p53 undergoes extensive multi-site phosphorylatio
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