Titlebook: Cell Engineering; Apoptosis Mohamed Al-Rubeai,Martin Fussenegger Book 2004 Springer Science+Business Media B.V. 2004 Mammalia.Regulation.Ti

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书目名称Cell Engineering影响因子(影响力)




书目名称Cell Engineering影响因子(影响力)学科排名




书目名称Cell Engineering网络公开度




书目名称Cell Engineering网络公开度学科排名




书目名称Cell Engineering被引频次




书目名称Cell Engineering被引频次学科排名




书目名称Cell Engineering年度引用




书目名称Cell Engineering年度引用学科排名




书目名称Cell Engineering读者反馈




书目名称Cell Engineering读者反馈学科排名

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978-90-481-6615-2Springer Science+Business Media B.V. 2004

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https://doi.org/10.1007/978-981-16-5613-2 chemical basis of these interactions is a major key to understanding the mechanisms of this diverse pathway, while controlling the outcome of these interactions offers remarkable benefits to the treatment of many pathological diseases. It is now undisputed that the Bcl-2 proteins regulate the mitoc

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Teruhisa Tsuzuki,Tamie NakajimaF signalling mechanisms contribute to cell transformation, tumour progression and metastasis. Many questions remain to be answered as to how exactly the IGF system mediates its effects both in normal and tumour cells and how the IGF-1R interacting proteins and downstream signalling cascades are regu

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Tamie Nakajima,Keiko Nakamura,Akihiko Kondohvel pathways and regulators will be elucidated and new targets for intervention will become available. As discussed in this review, work in many laboratories has indicated that manipulating the apoptosis machinery can, indeed, result in suppression of apoptosis and an extension of the productive lif

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