Titlebook: Cardiomyopathy and Myocardial Biopsy; M. Kaltenbach,F. Loogen (Direktor der I. Med. Klin Conference proceedings 1978 Springer-Verlag Berli

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Ultrastructure of Left Atrial Myocardium in Patients With Mitral Valvular Diseasew studies have been made of the ultrastructural pathology of human atria [6–17]. Although to date investigations of the fine structure of normal human atrial myocardium [15, 18, 19] have been much less detailed than have those of atrial muscle of other mammals [20–23], there is general agreement tha

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Hypertrophic and Degenerative Changes of the Myocardium and the Influence of Ischemia During Cardiacigated in order to evaluate the tolerable duration of induced cardiac arrest during open-heart surgery. It was concluded that 45 min of ischemia resulted in a moderate degree of myocardial damage and a relatively fast recovery of the tissue structure during reperfusion of the heart. However, toleran

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Martin Billeter,Vladislav Orekhovof myocardial disease. During the past 20 years, great interest has been focused on other factors, especially hormones and toxins that may produce myocardial cell damage. In addition, we became aware that pathology of heart muscle may also be of familial nature, thus suggesting genetic origin.

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CHOIR

overbearing

Alphavirus-Based Expression SystemsM. In these patients, heart volumes were slightly or moderately increased, they showed marked ECG changes, enddiastolic volumes were slightly to moderately increased, and ejection fractions slightly reduced.

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Functional Materials Developed in IMRrial was studied for diagnostic reasons mostly by use of light microscopy, which revealed specific findings only in rare cases (e. g. sarcoidosis and myocarditis). Furthermore, publications about biopsies consisted primarily of case reports and information about technical problems or modifications of the original Konno bioptome [10, 12].

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Peter H. Reuter Ph.D.,Bryce A. Pardom either directly or indirectly by interfering with cell metabolism. Changes of the electrolyte content (e.g., potassium, sodium, calcium, and chloride) can decrease the efficiency of the myocardial cell. In addition, toxic agents may damage the endothelium of intramural vessels and consequently ind

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