Titlebook: Cannabinoids; Roger G. Pertwee Book 2005 Springer-Verlag Berlin Heidelberg 2005 Cannabinoid.Nervous System.cannabinoid mechanisms of pain

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https://doi.org/10.1007/978-2-8178-0206-0docannabinoids appear to act as retrograde signalling agents, reducing synaptic inputs onto the stimulated neuron in a highly selective and restricted manner. In this review we describe the cellular mechanisms underlying retrograde endocannabinoid signalling.

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The Biosynthesis, Fate and Pharmacological Properties of Endocannabinoids, health and disease, and (2) templates for the design of new therapeutic drugs. This chapter summarizes the progress achieved in this direction during the 12 years following the discovery of the first endocannabinoid.

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Retrograde Signalling by Endocannabinoids,docannabinoids appear to act as retrograde signalling agents, reducing synaptic inputs onto the stimulated neuron in a highly selective and restricted manner. In this review we describe the cellular mechanisms underlying retrograde endocannabinoid signalling.

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Cannabinoid Receptor Signaling, tissues. CB. and CB. receptors are members of the superfamily of seven-transmembrane-spanning (7-TM) receptors, having a protein structure defined by an array of seven membrane-spanning helices with intervening intracellular loops and a C-terminal domain that can associate with G proteins. Cannabin

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Modulators of Endocannabinoid Enzymic Hydrolysis and Membrane Transport,nactivation. The purpose of this review is to compile available data regarding three inactivation processes: fatty acid amide hydrolase, monoacylglycerol lipase, and cellular membrane transport. In particular, we have focused on mechanisms by which these processes are modulated. We describe the in v