Titlebook: Cancer Microenvironment and Therapeutic Implications; Tumor Pathophysiolog Gianfranco Baronzio,Gianfranco Fiorentini,Christop Book 2009 Spr

Decibel

Adrenal-Glands

978-90-481-8160-5Springer Science+Business Media B.V. 2009

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R. López de Mántaras,C. Sierra,J. Agustí foreign body or chemicals. In every case, there is a protein leak from the damaged capillaries. This results in increased oncotic pressure which is in turn responsible for the methylation of the PP2A phosphatase. The activation of PP2A results in the translocation of NF κB and the activation of sev

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A Structure for Epistemic Stateswever, not only are cancers heterogeneous, but also small subsets are endowed with the ability to initiate cancer formation and metastasis. The resistance of cancer initiating cells to current therapies may explain high relapse rates. Our inability to eradicate cancer may be due to misrecognition of

Brain-Imaging

https://doi.org/10.1007/978-3-642-77681-6reach target tissue at adequate concentration to perform its therapeutic effect. As reported by several authors, conventional cancer treatments do not achieve this aim, resulting in suboptimal activity and inability to eradicate tumor tissue. The reasons for this failure are multiple and partially d

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New Directions for Medical Educationssure. New multi-target tyrosine kinase inhibitors are new therapeutically options in solid tumours. They share the capacity of modulate the hypoxia inducible factor (HIF)-VEGF-VEGF receptor sequence that plays a predominant role in the development of solid tumours.

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Needed: A New Way to Train Doctorsmour neo-vasculature result in development of oxygen deprived areas. Hypoxic cells existing in this environment are resistant to radiation therapy. Numerous approaches have been developed to deal with this hypoxia-induced radioresistance. These include increasing oxygen delivery to tumours, chemical