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Titlebook: Calcium and Contractility; Smooth Muscle A. K. Grover,E. E. Daniel Book 1985 The Humana Press Inc. 1985 ATP.Calcium.Nucleotide.cell membran

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Smooth Muscle Subcellular Fractionation,il 1971 when the detailed method of isolation and characterization of plasma membrane and other subcellular fractions was published (.). Recently, a great deal of interest has been shown in the smooth muscle cell surface calcium uptake and transport (.) and hence there is a requirement for pure plasma membranes for such studies.
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Smooth Muscle Relaxants,calcium-channel blockers (elucidating the role of calcium in normal cell function and drug action). This chapter attempts to discuss current pharmacodynamics, pharmacokinetics, and clinical pharmacology of smooth muscle relaxants and to give the researcher, clinician, graduate and medical student basic information on these compounds.
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Book 1985nisms responsible for them? How does the regula­ tion of contractility occur directly at the level of the actomyosin activity? What role do gap junctions play in cell-to-cell coupling? What are the roles of cholinergic, adrenergic, peptidergic, and nonadrenergic noncholinergic interactions in calciu
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Calcium Compartments and Mobilization During Contraction of Smooth Muscle,d in Chapters 1 and 11, various intracellular structures are capable of accumulating Ca.. The mobilization of such intracellularly bound Ca. pools can cause an increase in myoplasmic free Ca., independent of Ca. entry. Of course, the steady-state level of myoplasmic free Ca. is determined not only b
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Studies on Skinned Fiber Preparations,le can provide only suggestive information regarding the contractile machinery itself. On the other extreme, studies of the contractile mechanism with isolated contractile proteins lack the structural integrity of the contractile and regulatory systems that could be of critical importance in the int
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Calcium-Handling Defects and Smooth Muscle Pathophysiology,cellularly sequestered Ca.. For the initiation and maintenance of the state of relaxation after each contraction, inhibition of Ca. influx or release would not be adequate in lowering cytoplasmic Ca. contraction to 10. . or less because of the residual inward leak of Ca. across the cell membrane. A
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