Titlebook: Calcium Antagonists; Mechanism of Action Nicholas Sperelakis,James B. Caulfield Book 1984 Martinus Nijhoff Publishing, Boston 1984 calcium

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Clinical Electrophysiology of the Calcium Antagonistsofound importance in the generation and propagation of electrical activity in the SA and AV nodes. Although, in both the transitional cells of the SA node and the N cells of the AV node, sodium ions have a role in the initial generation of the action potential it is calcium which determines to a gre

irritation

Mechanisms of the Beneficial Effects of Some Ca2+ Antagonists on the Ca2+-Paradox in Myocardiumum are far from clear. Some investigators have shown the protective effects of both verapamil and diltiazem against the Ca. -paradox related myocardial injury and have interpreted their results to mean that the massive Ca. -influx during Ca. -paradox is occurring through the slow calcium channels (1

MILL

Comparative Effects of Ca Slow Channel Blockers on the Hamster Hereditary Cardiomyopathytary cardiomyopathy without changing the overall course of the generalized polymyopathy that prevails in these animals (7,9). Until now, the effectiveness of verapamil in hindering the progression of skeletal muscle changes has remained unexplained except for the fact that presumably Ca slow channel

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https://doi.org/10.1007/978-3-642-52615-2l studies have provided considerable information about the influx of calcium through specific membrane pores, the transport mechanisms responsible for the extrusion of calcium are less well understood (2). It has been assumed that accumulation of calcium in injured cells reflects predominantly a pas

现实

https://doi.org/10.1007/978-3-642-52615-2ofound importance in the generation and propagation of electrical activity in the SA and AV nodes. Although, in both the transitional cells of the SA node and the N cells of the AV node, sodium ions have a role in the initial generation of the action potential it is calcium which determines to a gre

incite

,Über synthetisch-apriorische Urteile,um are far from clear. Some investigators have shown the protective effects of both verapamil and diltiazem against the Ca. -paradox related myocardial injury and have interpreted their results to mean that the massive Ca. -influx during Ca. -paradox is occurring through the slow calcium channels (1

d-limonene

https://doi.org/10.1007/978-3-663-00199-7tary cardiomyopathy without changing the overall course of the generalized polymyopathy that prevails in these animals (7,9). Until now, the effectiveness of verapamil in hindering the progression of skeletal muscle changes has remained unexplained except for the fact that presumably Ca slow channel

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