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Titlebook: CNS Neuroprotection; Frank W. Marcoux (Vice President, Biology),Dennis Book 2002 Springer-Verlag Berlin Heidelberg 2002 Central Nervous S

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https://doi.org/10.1007/978-3-662-06274-6Central Nervous System; Chemokine; Gentherapie; Nervous System; Neurodegenerative Disease; Neuropharmakol
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978-3-642-07625-1Springer-Verlag Berlin Heidelberg 2002
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Narrating the Past through Theatretroke remains open, and there are other possible disease targets yet to be explored. Ongoing delineation of the cellular and molecular underpinnings of excitotoxicity has led to the progressive unveiling of countermeasures, aimed at attenuating presynaptic glutamate release, postsynaptic receptor ac
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Introduction: A Decade under Construction, under normal circumstances, the blood-brain barrier (BBB) exists and the cerebral microvessels express few of the adhesion molecules required for leukocyte trafficking into the brain. But when the brain is injured, the BBB becomes compromised (. et al. 1996) and there is an upregulation of leukocyt
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Methode „Visionen und erste Schritte“ses are overwhelmed. The central nervous system is particularly sensitive to oxygen radical damage because of the high levels of polyunsaturated lipids present that, when peroxidized, compromise the integrity of neuronal cell membranes together with the specialized functions of those membranes.
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