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派博传思国际中心 »期刊书目

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› BOOKS with Alphabet C (Ca, Cb,Cc, Cd, Ce…... )

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Titlebook: C-Myc in B-Cell Neoplasia; 14th Workshop on Mec Michael Potter (Chief),Fritz Melchers (Director) Book 1997 The Editor(s) (if applicable) an

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书目名称C-Myc in B-Cell Neoplasia
副标题14th Workshop on Mec
编辑Michael Potter (Chief),Fritz Melchers (Director)
视频videohttp://file.papertrans.cn/221/220083/220083.mp4
丛书名称Current Topics in Microbiology and Immunology
图书封面Titlebook: C-Myc in B-Cell Neoplasia; 14th Workshop on Mec Michael Potter (Chief),Fritz Melchers (Director) Book 1997 The Editor(s) (if applicable) an
描述The papers in this book were presented at the 14th Mechanisms in B-cell Neoplasia meeting that was held in Bethesda, Maryland October 21-23, 1996. In 1995 the organizers decided that the format of the meeting would be changed and that specific topics relevant to B-cell neoplasia would be discussed. This year‘s topic is on the c-myc oncogene in B-cell neoplasia which has been discussed in virtually every previous meeting. Some of the presentations announced for the first time dramatic advances in our understanding of c-myc and because this subject has become highly complex it was thought that devoting the whole meeting to this theme would be appropriate. The book, therefore, repre­ sents a review of many aspects of the myc problem but by no means is truly comprehensive. In a recent Medline search there were 8,505 references to myc, fully illustrating the magnitude of the interest and depth of this field. The organizers of the meeting have each contributed review chapters that summarize different aspects of the meeting. We thank the National Cancer Institute for sponsoring this workshop and the staff of Cygnus, Inc. , for their outstanding organizational assistance. The organizers ar
出版日期Book 1997
关键词DNA; apoptosis; cell; cytokine; growth; immunoglobulin; interferon; lymphoma; pathogenesis; protein; proteins;
版次1
doihttps://doi.org/10.1007/978-3-642-60801-8
isbn_softcover978-3-642-64560-0
isbn_ebook978-3-642-60801-8Series ISSN 0070-217X Series E-ISSN 2196-9965
issn_series 0070-217X
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
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B-lymphocyte-lineage Cells from Early Precursors to Ig-secreting Plasma Cells: Targets of Regulation miz-1 (see papers in this volume, in particular Austen et al., Schneider et al., Schreiber-Agus et al.), is thought to control expression of genes involved in the proliferation and differentiation of cells. Very few lineages of cellular development have been studied . and . in comparable detail as
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DNA Conformation, Topology, and the Regulation of , Expressionultitude of physiological processes and has been reported to be regulated by a long list of hormones, cytokines, lymphokines, nutritional status, development and differentiation. Myc levels can be pharmacologically perturbed, at least transiently, by a host of agents. Consistent with the notion that
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Structure and Expression of the c-Myc/Pvt 1 Megagene Locusing Burkitt’s Lymphoma (BL), AIDs-NHL, mouse plasmacytoma (Pct) and possibly multiple myeloma (MM). There is a restriction associated with this Tx such that only the immunoglobulin (Ig) heavy chain gene is found juxtaposed to c-Myc and only the Ig light chain gene is found juxtaposed to Pvt 1. Over
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c-Myc Promotes Survival of WEHI 231 B Lymphoma Cells from Apoptosisssion or inappropriate time of expression of the c-. gene has been found to promote apoptosis. Cleveland and coworkers observed that addition of a vector expressing c-Myc protein accelerated apoptosis following IL-3 deprivation of the 32D Independent myeloid cell line [3]. Similarly Evan and coworke
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Role of c-myc and p27 in Anti-IgM Induced B-Lymphoma Apoptosiso undetectable levels by 8–24 hours. These cells die soon thereafter via apoptosis. IgD receptor crosslinking also leads to an increase in c-myc expression, but it remains above baseline levels for more than 24 hours; these cells continue to proliferate and do not die. We previously reported that an
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