Titlebook: Atrial Fibrillation, a Treatable Disease?; J. H. Kingma,N. M. Hemel,K. I. Lie Book 1992 Springer Science+Business Media Dordrecht 1992 Abl

吹牛者

From Experiment to Therapeutic Innovation in Atrial Fibrillation and Fluttersion illustrates very well this disparity: ventricular fibrillation and tachycardia are the case in animal experiments, whereas accelerated idioventricular rhythm is today recognized a marker of reperfusion following thrombolysis in human coronary arteries[2,3].

戏法

The Pathology of Drug Resistant Lone Atrial Fibrillation in Eleven Surgically Treated Patientsl Heart Disease or of metabolic diseases. The pathological findings suggest that cardiomyopathic changes in the atria could be the anatomical substrate for the fibrillation. We report pathological findings in eleven cases of long-standing, drug-resistant AFs treated surgically with the . operation[12–15].

surmount

Termination of Atrial Fibrillation by Class IC Antiarrhythmic Drugs, a Paradox? it encounters tissue in a more or less advanced state of recovery. It may divide again or combine with a neighbor; it may be expected to fluctuate in size and change in direction. Its course, though determined by the excitability or refractoriness of surrounding tissue, would appear to be as random as Brownian motion”[2].

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0166-9842 l fibrillation than ever before. Also, there is a growing bodyof knowledge of the pathophysiologic mechanism, the pathology andepidemiology, and especially of the thrombo-embolic complications ofthis arrhythmia, which is responsible for a renewed interest of theclinician in this very common human ar

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peptic-ulcer

Episodic Treatment of Paroxysmal Atrial Fibrillationts quality of life. How to cope with the sudden onset of an attack is not only uncomfortable for the patient but a source of anxiety and concern over the ability to continue to function as usual. However, there is the old cliché: “You will have to live with it”.

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An AICD for Atrial Fibrillation?imals in whom the arrhythmia was initiated by topical application of acetyl choline and gentle traction of the atrial appendage. In the same model, 40–100 joules were needed for cardioversion with transthoracic shock. These findings relating to the success of low energy defibrillation have been corroborated by a number of other investigators[2,3].

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