Titlebook: Atrial Fibrillation; John Rawles Book 1992 Springer-Verlag London Limited 1992 Herzkrankheit.anatomy.arrhythmia.atrial fibrillation.cardia

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In Hilberts Hotel ist immer ein Zimmer frei,ce of narrow peaks on the histogram of R-R intervals; a relatively long atrioventricular conduction interval in sinus rhythm; rapid change of atrioventricular delay with change of ventricular rate; and a negative relation between cycle length of conducted impulses and the effective refractory period

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,Aufhören, wenn es am schönsten ist?,des useful information about cardiac function. Provided the circulation is adequately filled, a low stroke output indicates reduced cardiac function, while an above normal stroke output is attributable to increased circulatory volume and filling pressure. A normal stroke output may result from the c

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Models of the Atrioventricular Node,ce of narrow peaks on the histogram of R-R intervals; a relatively long atrioventricular conduction interval in sinus rhythm; rapid change of atrioventricular delay with change of ventricular rate; and a negative relation between cycle length of conducted impulses and the effective refractory period

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The Measurement of Cardiac Output in Sinus Rhythm and Atrial Fibrillation,des useful information about cardiac function. Provided the circulation is adequately filled, a low stroke output indicates reduced cardiac function, while an above normal stroke output is attributable to increased circulatory volume and filling pressure. A normal stroke output may result from the c

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The Management of Atrial Fibrillation,lls (Hertzeanu and Aron 1985). In fact, patients with atrial fibrillation have improved tolerance to orthostatic stress, and are less likely to suffer from postural hypotension (Abelman and Fareeduddin 1969). Syncope, or near- syncope, when it does occur in patients with atrial fibrillation is assoc

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The Pathophysiology of Atrial Fibrillation,, and the region where they are contiguous (James 1982; Ih and Saitoh 1982). Patchy, focal degeneration and fibrosis of atrial myocardium and the conducting system are characteristic, and predispose to atrial fibrillation by virtue of the resulting inhomogeneity of refractoriness and repolarisation.

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