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Titlebook: Atherosclerotic Plaques; Advances in Imaging Robert W. Wissler,M. Gene Bond,Gertrud Friedman Book 1991 Plenum Press, New York 1991 Dilatat

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https://doi.org/10.1007/978-3-030-57484-0s are acquiring the power and the resolution that may permit accurate assessment of plaque size and shape; of spatial disposition of lipid, mineral, and tissue components within a plaque; and of structural distortions and adaptations in the artery wall adjacent to an atherosclerotic lesion.
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Book 1991e scientists of different backgrounds and documented accomplishments together in a cloistered place for a few days to consider a very important and timely topic, many new ideas will be generated. The keynote of this conference was the Future. By this we mean the expected future developments of highl
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Open Source Software: Source of Innovation?, and diseased vessel wall has increased greatly. Some of these new developments are considered in order to understand better the behavior of atherosclerotic plaque during laser application and to begin to evaluate the possible uses of this powerful tool to alter and to measure components of the artery wall lesions using exogenous fluorescence.
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https://doi.org/10.1057/978-1-349-94854-3tle data on the physico-chemical structure of pathological alteration. The availability of a non-invasive method for the detection of morphological and physico-chemical features of atherosclerotic lesions and, possibly, the observation of their progression or regression, would represent a significant advance.
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How an Understanding of HDL’s Metabolism May Help us Discover New Ways to Evaluate Atherosclerosis ament (4), agreed on 35 mg/dl HDL cholesterol as a provisional cutoff value for risk assessment. This cutoff corresponds to the 16th percentile of the HDL cholesterol distribution in adult males in West Germany (Figure 2). Plasma concentrations below this cutoff may be associated with increased risk for premature onset of atherosclerosis.
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https://doi.org/10.1007/978-3-0348-9116-5al characterization of the fibroatheromatous plaques and their histogenesis are, however, still to be defined. Factors responsible for the evolution of intimal lesions and the mechanisms and stages of fibroatheromatous plaque formation are still largely obscure.
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Nitric Oxide and Related Radicalsxtent and composition by ultrasound in large and medium arteries: aortas and carotid arteries were both studied “in vitro” and “in vivo” by B-mode ultrasonographic devices and histopathology, often making use of endarteretomy samples from the carotid arteries.
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