Titlebook: Ataxia-Telangiectasia; Richard A. Gatti,Robert B. Painter Conference proceedings 1993 Springer-Verlag Berlin Heidelberg 1993 Ataxia-Telang

热心助人

Frankreichs Verfassung 1958–2008t approximately 5% of breast cancers may be the result of a highly penetrant autosomal dominant gene (Claus ., 1991). Such individuals tend to develop breast cancer at an early age, have a high prevalence of bilaterality and are at an elevated risk of other neoplasms (Lynch and Hirayam, 1989). Recen

NOMAD

增减字母法

Das Parteiensystem der V. Republikecificity. The mechanisms remain undefined. The physical basis for the differential accessibility in this site-specific recombination reaction has been a matter of much speculation. Possibilities have included the processes of transcription (Ferrier et al., 1989; Martin et al., 1991; Schlissel et al

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SIT

Nato ASI Subseries H:http://image.papertrans.cn/b/image/163910.jpg

临时抱佛脚

Isolation of Human cDNAS That Complement the Ataxia-Telangiectasia Phenotype in Cultured Fibroblastsgic degeneration as well as for cellular responses to induced and spontaneous DNA damage. However, despite extensive investigation, no A-T gene has been identified to date and the site of action of the A-T gene product(s) is unknown.

正论

AT-like Radiosensitive Rodent Cell Mutants: An Alternative Approach to the Isolation of the A-T Geneectasia, immunodeficiency, hypersensitivity to ionizing radiation and predisposition to cancer (for review see Sedgwick and Boder, 1991). Despite extensive investigation, the molecular defect responsible for these pleiotropic abnormalities in A-T remains unknown.

Terrace

Ataxia-Telangiectasia: A Brief Historical Overview1963.. By 1963 it was possible to publish a review of 101 cases world-wide illustrating the stereotyped nature of the disorder: progressive cerebellar ataxia in childhood, autosomal recessive heredity, oculocutaneous telangiectasia, frequency of sinopulmonary infection, peculiarity of eye movements,

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