Titlebook: Arterial Lesions and Arteriosclerosis; H. Jellinek (Professor of Pathology, Director) Book 1974 Akadémiai Kiadó, Budapest 1974 Lipoprotein

Dendritic-Cells

The Fine Structure of the Mammalian Arterial Wall, Bloom, 1957; Fawcett, 1959; Abramson, 1962; Bader, 1963; Buck, 1963; Hess and Stäubli, 1963., .; Lang, 1965; French, 1966). The following chapters deal only with those details which are essential to the understanding of the subject of this monograph.

保守

增长

商店街

发誓放弃

含糊

RADE

,Herstellung des Frischmörtels,ologic phenomena elicited by vascular impairment. Similar investigations have been carried out on muscular-type large vessels, using the same model of impairment by painting with acid. A total of 31 rabbits of both sexes, 2–3 years old and weighing on an average 4–5 kg, were used. A 3 cm long portio

dry-eye

contradict

Alexander Nikuradse,Karl Kuglernd Gorácz, 1954). The first light-microscopic change was the swelling and clumping (Altschul, 1957) of endothelial cells. Simultaneously, the smooth muscle cells of the media became swollen and vacuolized, and disseminated necroses appeared in the outer muscular layer of the media (Plate LXIII/45–49

debris

Alexander Nikuradse,Karl Kuglerf damage. This chapter deals with the further development of acute lesions. Studies have shown that the nature of the causative factor plays no role in the chronic stage of the change, but that the severity of the damage is decisive, because this determines both the morphology and the course of the