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Titlebook: Apoptosis; Enrico Mihich,Robert T. Schimke Book 1994 Springer Science+Business Media New York 1994 Antigen.DNA.apoptosis.cancer.cell.cell

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https://doi.org/10.1007/978-3-322-85163-5hat this recognition prompted the appearance of models of immune tolerance, and served as the impetus for the development of the clonal selection theory (2,3). The application of this theory to explain tolerance through the deletion of potentially autoreactive cells as proposed by Burnet (3,4) persist in almost unchanged form today.
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,: Oncogene and Tumour Suppressor Gene,, however, extremely rare as demonstrated by the fact that cancer arises in only one in three individuals during the entire course of their lives. Part of the answer to this paradox appears to reside in the multifunctional nature of the components that mediate cell proliferation. One of these, c-Myc, is the subject of this paper.
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Ion Signalling in Apoptosis,1992), whereas viruses (Rao et al., 1992) and bacterial toxins (Fiorentini et al, 1993) can directly elicit an apoptotic response in target cells. Finally, exposure to ionizing radiation (Zhivotovsky et al., 1981 and 1993b) or chemical toxicants (McConkey et al., 1988; Aw et al., 1990) can accelerate apoptosis in immature T cells.
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MYC, FAS, Apoptosis, and Immune Tolerance,hat this recognition prompted the appearance of models of immune tolerance, and served as the impetus for the development of the clonal selection theory (2,3). The application of this theory to explain tolerance through the deletion of potentially autoreactive cells as proposed by Burnet (3,4) persist in almost unchanged form today.
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978-1-4757-9219-5Springer Science+Business Media New York 1994
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