Titlebook: A Multidisciplinary Approach to Myelin Diseases II; S. Salvati Book 1994 Springer Science+Business Media New York 1994 Antigen.biology.bra

septicemia

Retrovirus-Mediated Gene Transfer of PMP22 in Schwann Cells: Studies on Cell Growthpolypeptides that are present both in normal and transformed cells and that are common to many cell types analyzed (9). Some of these polypeptides may correspond to . (12–14; see also other articles in this volume), and the elucidation of their function may lead a better understanding of the process

偶然

Protein Sorting and Targeting in Myelin-Forming Schwann Cellsc material — the breaking and making of internucleotide bonds or the modification of DNA nucleotides — and not the passive use of DNA as a template, as in transcription (Chapter 2). These processes, DNA replication, recombination and repair (mutation has been discussed in Chapter 3), have many gene

性满足

Angioplasty

西瓜

Mechanisms of DE — and Remyelination in Autoimmune Encephalomyelitis and Multiple Sclerosisof these proteins, ovalbumin, has been studied extensively. Ovalbumin mRNA has been purified (Rosen et al. 1975), and a full-length dsDNA copy synthesized (Monahan et al. 1976b) and cloned in a bacterial plasmid (McReynolds et al. 1977). More recently, ovalbumin genomic DNA sequences have been isola

dendrites

The Immunologic Response of the Oligodendrocyte in the Active Multiple Sclerosis Lesionrest in the structure of chromatin beyond the nucleosome-that is, the superstructural characteris­ tics of the genetic material-is finally yielding meaningful results that give promise for understanding the regulation of gene activity. ROBERT F. GOLDBERGER Preface Research on the molecular mechanism

母猪

可耕种

Intrathecal Cytokine Synthesis in Inflammatory and Demyelinating Diseases of the Central Nervous Sysors of lactotropes (GH., PRL.), the last cell type to appear along this lineage (Hoeffler et al., 1985; Behringer et al., 1988). Various data indicate the existence of a subpopulation of GH. cells which can switch to PRL. cells and PRL. cells which can switch to GH. cells, under the influence of cer

心神不宁

Lineages, Cell Fate Determination, and Migration in CNS Gliogenesisthe promoter and intron 1 leads to over-expression of . mRNA and an imbalance in production of the collagen type 1 α1 chain relative to the α2 chain. Polymorphisms in the regulatory regions of . and . have also been described which modulate gene expression, but the mechanisms by which these predispo

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