包庇
发表于 2025-3-26 21:48:39
Mitsuru Emi,Tsunenori Hirayama,Masahiro Tsuji,Akira Hata
PAD416
发表于 2025-3-27 02:42:58
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变化
发表于 2025-3-27 08:03:54
Paivi Pajukanta,Christian Ehnholm,Marja-Riitta Taskinen,Leena Peltonen
SUE
发表于 2025-3-27 12:51:59
Toru Kita (Professor),Masayuki Yokode (Assistant PPresenting the exciting results of research in the areas of the molecular basis of lipoprotein metabolism,.the link between lipoprotein metabolism disorder and atherogenesis, and the molecular approac
令人心醉
发表于 2025-3-27 15:12:02
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Femine
发表于 2025-3-27 19:46:06
Molecular Analysis on the LDL Receptors in Two Patients with Homozygous Familial Hypercholesterolemiint mutation in exon 7 and the other is a replacement of nine bases with completely different six bases in exon 8. Patient KZA is a true homozygote for a splice donor site mutation in intron 11. As expected from the sites of mutations, Patient NS retained some LDL receptor activity whereas Patient KZA showed no activity.
Enrage
发表于 2025-3-27 22:29:35
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seduce
发表于 2025-3-28 03:49:29
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Ascribe
发表于 2025-3-28 07:34:40
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Debark
发表于 2025-3-28 11:27:26
Common Mutation of 5, 10-Methylenetetrahydrofolate Reductase Accelerates Coronary Artery Disease in s, only MTHFR genotype W was shown to be an independent predictor of the early onset of CAD. The mean plasma Hcy level of genotype W was significantly higher than those of the other two genotypes. These results suggest that he MTHFR mutation may accelerates the onset of CAD through elevation of plasma Hcy levels in male heterozygous FH patients.