万灵药 发表于 2025-3-21 17:43:47

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迫击炮 发表于 2025-3-21 23:09:12

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installment 发表于 2025-3-22 01:10:25

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善于 发表于 2025-3-22 06:16:13

Mechanism Regulating Recruitment of CD11b/CD18 to the Cell Surface is Distinct From That Which Induc8, the common 95 kD beta subunit (.2) abolish neutrophil aggregation (2); (ii) several of the MAb to CD11b, the 165 kD alpha subunit (also referred to as αM, CR3, or MAC-1 inhibit this response (2,3); and (iii) neutrophils from individuals genetically deficient in these molecules cannot be stimulated to aggregate (4).

GROG 发表于 2025-3-22 10:25:58

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ungainly 发表于 2025-3-22 16:21:57

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PRO 发表于 2025-3-22 18:22:15

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floodgate 发表于 2025-3-22 22:57:07

ere antibodies against these molecules are given to inhibit their function. The blend of basic science and clinical applications provides clear evidence of the biological relevance of cell-cell interactions and the many potential clinical dividends afforded by understanding the molecular basis of cell adhesion.978-1-4612-7927-3978-1-4612-3234-6

fluffy 发表于 2025-3-23 05:20:53

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参考书目 发表于 2025-3-23 07:15:17

CD18 Dependence of Primate Eosinophil Adherence In Vitroedictive of an intense immediate response to inhaled antigen (6). Additionally, chronic airway eosinophilia in monkeys, either idiopathic (7) or induced by chronic antigen exposure (8), is associated with marked airway hyperresponsiveness to inhaled methacholine.
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查看完整版本: Titlebook: Leukocyte Adhesion Molecules; Proceedings of the F Timothy A. Springer (Associate Professor),Donald C Conference proceedings 1990 Springer-