Meditate 发表于 2025-3-26 23:29:13

https://doi.org/10.1007/978-2-8178-0206-0docannabinoids appear to act as retrograde signalling agents, reducing synaptic inputs onto the stimulated neuron in a highly selective and restricted manner. In this review we describe the cellular mechanisms underlying retrograde endocannabinoid signalling.

最小 发表于 2025-3-27 03:33:47

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衍生 发表于 2025-3-27 07:24:03

The Biosynthesis, Fate and Pharmacological Properties of Endocannabinoids, health and disease, and (2) templates for the design of new therapeutic drugs. This chapter summarizes the progress achieved in this direction during the 12 years following the discovery of the first endocannabinoid.

debunk 发表于 2025-3-27 11:04:36

Retrograde Signalling by Endocannabinoids,docannabinoids appear to act as retrograde signalling agents, reducing synaptic inputs onto the stimulated neuron in a highly selective and restricted manner. In this review we describe the cellular mechanisms underlying retrograde endocannabinoid signalling.

Kindle 发表于 2025-3-27 17:09:09

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lacrimal-gland 发表于 2025-3-27 21:11:08

Cannabinoid Receptor Signaling, tissues. CB. and CB. receptors are members of the superfamily of seven-transmembrane-spanning (7-TM) receptors, having a protein structure defined by an array of seven membrane-spanning helices with intervening intracellular loops and a C-terminal domain that can associate with G proteins. Cannabin

积习已深 发表于 2025-3-27 23:17:05

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OTTER 发表于 2025-3-28 03:16:26

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Flirtatious 发表于 2025-3-28 06:52:25

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护航舰 发表于 2025-3-28 12:26:54

Modulators of Endocannabinoid Enzymic Hydrolysis and Membrane Transport,nactivation. The purpose of this review is to compile available data regarding three inactivation processes: fatty acid amide hydrolase, monoacylglycerol lipase, and cellular membrane transport. In particular, we have focused on mechanisms by which these processes are modulated. We describe the in v
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查看完整版本: Titlebook: Cannabinoids; Roger G. Pertwee Book 2005 Springer-Verlag Berlin Heidelberg 2005 Cannabinoid.Nervous System.cannabinoid mechanisms of pain