oxidation
发表于 2025-3-21 17:10:28
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凝视
发表于 2025-3-21 21:33:20
and immunity. Dietary sources can be converted into beneficial metabolic end-products such as short chain fatty acids, i.e., acetate, propionate, and butyrate, fermented by the beneficial gut microbiota such as . and .. This is achieved by an indirect nutrient strategy using pro/prebiotic. The gut
包庇
发表于 2025-3-22 02:50:39
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Encumber
发表于 2025-3-22 08:10:08
Thomas Laue,Andreas Plagensage and activate pro-inflammatory and pro-fibrotic pathways, thus potentially contributing to SSc pathogenesis. Since dismicrobism can be corrected with opportune hygienic-dietary and therapeutic interventions resulting in indirect benefits on disease course, the fascinating role of microorganisms i
REP
发表于 2025-3-22 10:44:02
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不能仁慈
发表于 2025-3-22 13:04:40
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广告
发表于 2025-3-22 18:00:24
Thomas Laue,Andreas Plagense system. The aim of this chapter is to summarize the main microbes studied and characterized so far in relation to AD and to outline their role in the complex pathogenetic mechanism of the disease and in the macrocosm represented by the AD patient, also in view of the possible current and future im
CBC471
发表于 2025-3-22 23:35:36
Thomas Laue,Andreas Plagensirus, herpes simplex virus 2 and Zika virus, as well as gut microbiome and NMOSD occurrence are mentioned. On the other hand, susceptibility of NMOSD patients for developing infectious diseases due to receiving immunosuppressive drugs and the role of infection in NMOSD attack and disease exacerbatio
irritation
发表于 2025-3-23 03:57:40
Thomas Laue,Andreas Plagenspecific sensitization by the body’s own reactive T-cells, axonal injury, and edema. As no specific etiologic agent has been constantly identified behind ADEM, and the role of any specific underlying mechanism is still debated, more research is needed to further establish the relationship with these
羊齿
发表于 2025-3-23 09:06:52
Thomas Laue,Andreas Plagensas Th-1 and Th-17 are produced in response to invasive gut microorganisms (e.g., TNF-α, IFN-γ) by enhancing the transcription of significant genes. This chapter focuses on all the alternative pathophysiology and pathogenesis and management related to ulcerative colitis.