强所 发表于 2025-3-25 03:24:55
John Leavittet al., 1990). When the gram-negative bacterium, . is treated with a heat shock, the cell responds by transiently accelerating the rate of transcription of heat shock genes. This increased transcription is promoted by the Eσ. RNA polymerase holoenzyme. The genes of approximately half of the 20 or soBlood-Clot 发表于 2025-3-25 10:11:35
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http://reply.papertrans.cn/59/5813/581212/581212_24.png认识 发表于 2025-3-25 22:30:55
Piotr Blumczynskihavior, hormone release, metabolism, and so on. Even in homeothermic mammals, body temperature fluctuates in a circadian manner that transmits the time information to peripheral tissues and cells. The body temperature rhythms cause cyclic activation of the transcription factor HSF1 and its targets s适宜 发表于 2025-3-26 01:41:55
http://reply.papertrans.cn/59/5813/581212/581212_26.png滑稽 发表于 2025-3-26 07:40:24
Taras Shmiherams and tables to facilitate readers’ systematic understandiThis book presents a large amount of information related to the heat shock response and heat shock factor (HSF), describes core observations about molecular mechanisms and pathophysiological roles, and provides fundamental concepts on the bBABY 发表于 2025-3-26 10:21:43
http://reply.papertrans.cn/59/5813/581212/581212_28.png令人苦恼 发表于 2025-3-26 15:42:26
(AIRDs), including rheumatoid arthritis (RA), systemic lupus erythematosus (SLE), systemic sclerosis, Sjogren’s syndrome and various idiopathic vasculitides. Anti-human Hsp60 autoantibodies can be detected in the sera of patients with AIRDs in various frequencies and levels; therefore, their diagnosJargon 发表于 2025-3-26 16:54:45
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