correspondent 发表于 2025-3-26 23:06:25
http://reply.papertrans.cn/48/4704/470396/470396_31.pngintoxicate 发表于 2025-3-27 05:03:44
http://reply.papertrans.cn/48/4704/470396/470396_32.pngalabaster 发表于 2025-3-27 05:49:35
http://reply.papertrans.cn/48/4704/470396/470396_33.pngGLIDE 发表于 2025-3-27 12:49:23
Opposite Modulation of Peripheral Inflammation and Neuroinflammation by Adenosine A2A Receptorsview the evidences suggesting that A2A receptor control neuroinflammation and discuss the possible mechanisms underlying the opposite modulation by A2A receptors of peripheral inflammation and neuroinflammation.OTHER 发表于 2025-3-27 16:55:45
http://reply.papertrans.cn/48/4704/470396/470396_35.png中子 发表于 2025-3-27 17:54:14
Inflammation and Neuronal Susceptibility to Excitotoxic Cell Deathlight into the cellular and molecular mechanisms of neurodegeneration. The response of microglia and astrocytes to injury, releasing signaling molecules (specially, cytokines, chemokines, growth factors, reactive oxygen and nitrogen species), is central in neuroinflammation and in neuron death/survival.针叶 发表于 2025-3-28 01:51:06
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http://reply.papertrans.cn/48/4704/470396/470396_38.pngObedient 发表于 2025-3-28 09:39:35
http://reply.papertrans.cn/48/4704/470396/470396_39.pngROOF 发表于 2025-3-28 14:00:19
Neuroinflammation and Excitotoxicity in Neurobiology of HIV-1 Infection and AIDS: Targets for Neuropical and inflammatory response involving all cell types in the brain. In fact, HIV-1 seems to enter the brain very soon after infection and can subsequently induce severe and debilitating neurological problems that range from mild behavioral abnormalities and motor dysfunction to frank dementia.