粘连 发表于 2025-3-25 05:49:47
Neuroinflammation and Mitochondrial Dysfunction in Alzheimer’s and Prion’s Diseasesmyloidogenic proteins, amyloid-beta (A ), and pathogenic scrapie prion (PrPSc), respectively. In both disorders, cerebral amyloid deposits are associated with a local inflammatory response, which is initiated by the activation of microglia and recruitment of astrocytes. Activated microglia, particul使混合 发表于 2025-3-25 08:08:26
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athology.Includes supplementary material: .Interactions Between Neurons and Glia in Aging and Disease is a source of "reference reviews" and "concise overviews" of the functional cross-talk between neurons and glial cells, with a focus on microglia and inflammation in the aging brain and in neurologInduction 发表于 2025-3-25 22:48:08
Book 2007tween neurons and glial cells, with a focus on microglia and inflammation in the aging brain and in neurological disorders. The scientific area covered by this book may be considered of key interest, and it is recognized, by the scientific community, as a major competitive area with critical implica上腭 发表于 2025-3-26 00:31:34
http://reply.papertrans.cn/48/4704/470396/470396_26.pngphase-2-enzyme 发表于 2025-3-26 06:37:04
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Aging and Cognitive Decline: Neuroprotective Strategiesy phenomena in dementia, especially in Alzheimer’s disease (AD), is impressive, and the role of inflammatory cells and mediators has been extensively studied. Furthermore, anti-inflammatory drugs (specially the nonsteroidal anti-inflammatory drugs (NSAIDs)) have been shown to attenuate these phenomena in preclinical studies.虚度 发表于 2025-3-26 13:01:17
Neuroinflammation and Mitochondrial Dysfunction in Alzheimer’s and Prion’s Diseasesarly those in the vicinity of amyloid deposits can produce and release proinflammatory cytokines, chemokines, complement proteins, acute-phase proteins, and reactive oxygen and nitrogen species that can damage the neighboring neurons.EXUDE 发表于 2025-3-26 20:34:42
The Impact of an Imbalance Between Proinflammatory and Anti-inflammatory Influences on Synaptic funct it is an imbalance between proinflammatory and anti-inflammatory cytokines that leads to age-related deficits in synaptic function and that strategies that restore the balance are likely to be beneficial in reducing the deterioration of function that accompanies age.