Boldfaced 发表于 2025-3-21 18:00:03

书目名称G Proteins, Receptors, and Disease影响因子(影响力)<br>        http://figure.impactfactor.cn/if/?ISSN=BK0380015<br><br>        <br><br>书目名称G Proteins, Receptors, and Disease影响因子(影响力)学科排名<br>        http://figure.impactfactor.cn/ifr/?ISSN=BK0380015<br><br>        <br><br>书目名称G Proteins, Receptors, and Disease网络公开度<br>        http://figure.impactfactor.cn/at/?ISSN=BK0380015<br><br>        <br><br>书目名称G Proteins, Receptors, and Disease网络公开度学科排名<br>        http://figure.impactfactor.cn/atr/?ISSN=BK0380015<br><br>        <br><br>书目名称G Proteins, Receptors, and Disease被引频次<br>        http://figure.impactfactor.cn/tc/?ISSN=BK0380015<br><br>        <br><br>书目名称G Proteins, Receptors, and Disease被引频次学科排名<br>        http://figure.impactfactor.cn/tcr/?ISSN=BK0380015<br><br>        <br><br>书目名称G Proteins, Receptors, and Disease年度引用<br>        http://figure.impactfactor.cn/ii/?ISSN=BK0380015<br><br>        <br><br>书目名称G Proteins, Receptors, and Disease年度引用学科排名<br>        http://figure.impactfactor.cn/iir/?ISSN=BK0380015<br><br>        <br><br>书目名称G Proteins, Receptors, and Disease读者反馈<br>        http://figure.impactfactor.cn/5y/?ISSN=BK0380015<br><br>        <br><br>书目名称G Proteins, Receptors, and Disease读者反馈学科排名<br>        http://figure.impactfactor.cn/5yr/?ISSN=BK0380015<br><br>        <br><br>

Haphazard 发表于 2025-3-22 00:07:23

Albright Hereditary Osteodystrophy, Pseudohypoparathyroidism, and Gs Deficiency, characterized by hypocalcemia, hyperphosphatemia, and elevation of serum PTH in the setting of normal renal function, is the major clinical feature. In the original report by Fuller Albright, PHP patients showed reduced calcemic and phosphaturic responses to injected bovine parathyroid extract comp

opportune 发表于 2025-3-22 04:18:23

,Gαs-Activating Mutations,ot only in all vertebrates, but also in invertebrates, such as . and .. G. couples receptors for numerous peptide hormones and monoamines to stimulation of adenylyl cyclase. Although it is now clear that G-protein βγ-subunits can independently modulate adenylyl cyclase (positively or negatively depe

arthrodesis 发表于 2025-3-22 06:28:36

http://reply.papertrans.cn/39/3801/380015/380015_4.png

形容词词尾 发表于 2025-3-22 12:14:31

http://reply.papertrans.cn/39/3801/380015/380015_5.png

厚脸皮 发表于 2025-3-22 13:08:16

,Gα12- and Gα13-Subunits of Heterotrimeric G-Proteins A Novel Family of Oncogenes,receptors in normal and aberrant cell growth. As such, a number of oncogenes have been found to code for altered forms of these receptors ., their ligands (.), or for molecules thought to participate in their growth-promoting pathways .. Another family of structurally related cell-surface receptors

厚脸皮 发表于 2025-3-22 18:53:33

Hypo- and Hyperthyroidism Caused by Mutations of the TSH Receptor,imarily through a positive control exerted by the pituitary hormone thyroid stimulating hormone (TSH) and a negative control exerted by iodine. In most physiological circumstances the maintenance of eumetabolism requires steady concentrations of circulating thyroid hormones. This is achieved by a cl

parasite 发表于 2025-3-22 22:18:46

http://reply.papertrans.cn/39/3801/380015/380015_8.png

Malcontent 发表于 2025-3-23 02:56:01

Inactivating and Activating Mutations of the FSH Receptor Gene,that participate in the regulation of reproductive functions, and those of the gonadotropin receptors (R) have received considerable attention. Since the discovery of the first luteinizing hormone receptor (LHR) mutations in 1993 ., more than 15 mutations have been so far reported in this gene (. Ch

诽谤 发表于 2025-3-23 09:12:13

Nephrogenic Diabetes Insipidus and Vasopressin Receptor Mutations,g tubules contain selective cells known as principal cells, responsive to the neurohypophyseal antidiuretic hormone arginine-vasopressin (AVP). The major action of AVP is to facilitate urinary concentration by allowing water to be transported passively down an osmotic gradient between the tubular fl
页: [1] 2 3 4 5 6 7
查看完整版本: Titlebook: G Proteins, Receptors, and Disease; Allen M. Spiegel (Chief, Metabolic Diseases Branch Book 1998 Springer Science+Business Media New York