incredulity 发表于 2025-3-25 06:19:01

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诱骗 发表于 2025-3-25 09:28:41

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NORM 发表于 2025-3-25 12:26:01

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somnambulism 发表于 2025-3-25 16:35:30

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乞讨 发表于 2025-3-25 21:53:49

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farewell 发表于 2025-3-26 04:11:36

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评论者 发表于 2025-3-26 05:47:53

Nephrogenic Diabetes Insipidus and Vasopressin Receptor Mutations,jor action of AVP is to facilitate urinary concentration by allowing water to be transported passively down an osmotic gradient between the tubular fluid and the surrounding interstitium. The process of this counter multiplication system and the action of AVP on principal collecting duct cells are represented in Fig. 1.

门窗的侧柱 发表于 2025-3-26 09:08:01

https://doi.org/10.1007/978-3-658-05955-2t hypercal-cemia, is caused by activating mutations in the parathyroid hormone (PTH) PTH-related peptide (PTHrP) receptor. These findings are likely to have significant implications for understanding the biological importance of the PTH/PTHrP receptor in addition to its role in regulating mineral ion homeostasis.

修正案 发表于 2025-3-26 15:17:24

,Constitutively Active PTH/PTHrP Receptors Cause Jansen’s Metaphyseal Chondrodysplasia,t hypercal-cemia, is caused by activating mutations in the parathyroid hormone (PTH) PTH-related peptide (PTHrP) receptor. These findings are likely to have significant implications for understanding the biological importance of the PTH/PTHrP receptor in addition to its role in regulating mineral ion homeostasis.

Factorable 发表于 2025-3-26 17:23:22

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查看完整版本: Titlebook: G Proteins, Receptors, and Disease; Allen M. Spiegel (Chief, Metabolic Diseases Branch Book 1998 Springer Science+Business Media New York