staging 发表于 2025-3-25 04:47:53
The Efferent Arm of the Energy Balance Regulatory Pathway: Neuroendocrinology and Pathology,rough afferent neural and hormonal signals reaching the hypothalamus, with resultant efferent projections of the autonomic nervous system innervating the muscles, viscera, and adipose tissue. The efferent system is of particular interest, as it has its own pathologies, which can be modulated to achi流逝 发表于 2025-3-25 10:49:45
http://reply.papertrans.cn/32/3104/310334/310334_22.pnghandle 发表于 2025-3-25 14:05:27
Interaction Between Physical Activity and Genetic Factors in Complex Metabolic Disease,essed carbohydrates is likely to have increased and physical activity levels declined. However, the rates at which obesity and diabetes have increased differ across people of varying ethnicities living in the same environment, suggesting the presence of interaction between ethnic-specific factors, s宇宙你 发表于 2025-3-25 19:43:51
http://reply.papertrans.cn/32/3104/310334/310334_24.pngJogging 发表于 2025-3-25 23:06:15
,Antipsychotic Medication–Induced Weight Gain and Risk for Diabetes and Cardiovascular Disease, and cardiovascular disease (CVD), with related increases in mortality. Increased adiposity is associated with decreases in insulin sensitivity, leading to increased risk of hyperglycemia and hyperlipidemia. Current evidence supports the hypothesis that treatment with antipsychotic medications is asirradicable 发表于 2025-3-26 04:01:02
The Surgical Approach to Morbid Obesity,inning in 1966, which restricts food intake and produces less malabsorption. Both operations exposed the distal ileum to glucose and fat, which stimulates secretion of glucagon-like peptide-1 (GLP-1). This is a brake hormone for gastric emptying and intestinal peristalsis. GLP-1 is also an incretinmaudtin 发表于 2025-3-26 06:27:06
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Zulejha Merhemic,Majda M. Thurnhertaining normal body weight in humans is highlighted by the finding that disordered melanocortin signaling results in early-onset morbid obesity and dramatic increases in lean body mass in humans. Emerging evidence suggests that blocking this system via pharmacologic antagonists to the type 4 melanoc