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发表于 2025-3-21 16:40:47
书目名称Discoidin Domain Receptors in Health and Disease影响因子(影响力)<br> http://figure.impactfactor.cn/if/?ISSN=BK0280916<br><br> <br><br>书目名称Discoidin Domain Receptors in Health and Disease影响因子(影响力)学科排名<br> http://figure.impactfactor.cn/ifr/?ISSN=BK0280916<br><br> <br><br>书目名称Discoidin Domain Receptors in Health and Disease网络公开度<br> http://figure.impactfactor.cn/at/?ISSN=BK0280916<br><br> <br><br>书目名称Discoidin Domain Receptors in Health and Disease网络公开度学科排名<br> http://figure.impactfactor.cn/atr/?ISSN=BK0280916<br><br> <br><br>书目名称Discoidin Domain Receptors in Health and Disease被引频次<br> http://figure.impactfactor.cn/tc/?ISSN=BK0280916<br><br> <br><br>书目名称Discoidin Domain Receptors in Health and Disease被引频次学科排名<br> http://figure.impactfactor.cn/tcr/?ISSN=BK0280916<br><br> <br><br>书目名称Discoidin Domain Receptors in Health and Disease年度引用<br> http://figure.impactfactor.cn/ii/?ISSN=BK0280916<br><br> <br><br>书目名称Discoidin Domain Receptors in Health and Disease年度引用学科排名<br> http://figure.impactfactor.cn/iir/?ISSN=BK0280916<br><br> <br><br>书目名称Discoidin Domain Receptors in Health and Disease读者反馈<br> http://figure.impactfactor.cn/5y/?ISSN=BK0280916<br><br> <br><br>书目名称Discoidin Domain Receptors in Health and Disease读者反馈学科排名<br> http://figure.impactfactor.cn/5yr/?ISSN=BK0280916<br><br> <br><br>
构成
发表于 2025-3-21 22:39:31
Staging and Performing Translationnd implantation, as well as wound healing and auditory sensation. In addition, genetically modified mice and DDR mutant mice highlight the role of DDRs in the enhancement or attenuation of diseases, including fibrosis, atherosclerosis, and osteoarthritis in various conditions..Studies on mouse model
合并
发表于 2025-3-22 03:02:48
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单调女
发表于 2025-3-22 06:05:59
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Nebulizer
发表于 2025-3-22 09:24:04
https://doi.org/10.1007/978-3-322-94744-4etastasis. The molecular mechanisms underlying how DDRs contribute to these and other pathologies need to be understood to find new markers and for development of therapeutic agents for treatment of disease. This is particularly the case for EOC in which mechanisms explaining the atypically high lev
Demonstrate
发表于 2025-3-22 13:30:50
https://doi.org/10.1007/978-3-322-93990-6 we describe the key molecular interactions and signalling pathways elucidated by these studies and where appropriate highlight situations where signalling outcomes appear to be dependent on cellular context. We present an emerging molecular portrait of the DDRs and highlight areas where more intens
Demonstrate
发表于 2025-3-22 20:15:20
https://doi.org/10.1007/978-3-322-93990-6llagen-independent cell–cell interactions. Collagen-activated DDR1 signals through NF-kB, PI3K/Akt and p38, JNK, and ERK1/2 MAPKs, while inactive DDR1 appears to interact with E-cadherin promoting cell–cell interactions. DDR1 interacts with several other receptors, including Notch1 and Frizzled5, an
ostrish
发表于 2025-3-22 21:38:41
https://doi.org/10.1007/978-3-663-01602-1n osteoarthritic joint. If, as we suspect, DDR2 is one of the major contributors to progressive joint failure, then drugs that inhibit the kinase activity of DDR2 may be able to ameliorate osteoarthritis conditions.
和平主义者
发表于 2025-3-23 01:53:10
https://doi.org/10.1007/978-3-663-01602-1e pathways were not activated which resulted to an impressive preservation of renal function and structure. Further proof of evidence came from experiments with in vivo administration of antisense oligonucleotides against DDR1. The fact that this pharmacogenetic approach protected animals against th
人类
发表于 2025-3-23 06:46:42
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