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978-1-4684-9844-8Springer Science+Business Media New York 2004
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KangWoo Lee,Jung-Hoon Hwang,Dong-Soo Kwontibility complex (MHC) molecules. However, the TCR : MHC interaction is insufficient to trigger productive activation of T cells. It is now widely recognized that a second signal known as costimulation is required for the productive activation of antigen (Ag)-specific T cells.
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Accentuating Tumor Immunity Through Costimulationtibility complex (MHC) molecules. However, the TCR : MHC interaction is insufficient to trigger productive activation of T cells. It is now widely recognized that a second signal known as costimulation is required for the productive activation of antigen (Ag)-specific T cells.
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Aineas Mallios,Ted Lindblom,Stefan Sjögrennate the malignant cells and the chronic infectious agent. This setting is not uncommon in mice or patients with a congenital or acquired immunodeficiencies. However, this problem is also frequently seen in fully immunocompetent individuals, suggesting that malignant cells and certain microorganisms
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Kristaps Freimanis,Maija Šenfelded proliferation of different immune cells. Naïve T cells are unable to directly recognize tumor antigens. They require help from specialized cells known as professional antigen-presenting cells (APCs). APCs first interact with tumors, uptake tumor cells/proteins, process them, and present antigenic