DRAFT 发表于 2025-3-28 15:21:27
http://reply.papertrans.cn/19/1892/189156/189156_41.pngcoagulation 发表于 2025-3-28 22:28:40
http://reply.papertrans.cn/19/1892/189156/189156_42.png放牧 发表于 2025-3-28 23:02:49
http://reply.papertrans.cn/19/1892/189156/189156_43.pngacetylcholine 发表于 2025-3-29 05:07:49
http://reply.papertrans.cn/19/1892/189156/189156_44.png描述 发表于 2025-3-29 11:07:29
Design und Auswertung einer Anwendungsstudieclude specialized laminins, collagens, integrins, as well as other unique structural components which together serve to connect the intermediate filament network of the basal keratinocyte cytoskeleton to the interstitial collagen network of the papillary dermis. Damage to the dermal-epidermal basemecharisma 发表于 2025-3-29 12:15:13
http://reply.papertrans.cn/19/1892/189156/189156_46.pngpacific 发表于 2025-3-29 16:11:01
Design und Auswertung einer Anwendungsstudieave been shown to regulate additional biological processes of importance such as protein synthesis, cell migration, and apoptosis. Much of this knowledge has been gained through the study of rare disorders, known as keratinopathies, which are caused by mutations in genes encoding the various epiderm标准 发表于 2025-3-29 22:45:37
Öffentlicher Raum als privater Bildungsraumosome anchoring complexes. Over the last few years, however, the clinicopathological spectrum of EB has been expanded to also include primary abnormalities in desmosome junctions. Desmosomes are intercellular junctions that contribute to cell-cell adhesion, signalling, development and differentiatiointrude 发表于 2025-3-30 03:53:01
Wolfgang Wittwer,Andreas Diettrichere are several variants of pemphigus, each with unique clinical, histological, and immunologic features. Interestingly, when different desmosomal proteins are targeted by the autoimmune response, different clinical and histological features are seen. In this chapter we review the key intercellularToxoid-Vaccines 发表于 2025-3-30 04:50:46
Wolfgang Wittwer,Andreas Diettrichthelial cells to the underlying basement membrane. It is a homotrimeric type II transmembrane protein consisting of three 180 kDa alpha-1 (XVII) chains. Each individual chain is encoded by the . gene. Mutations in . usually lead to loss of collagen XVII and a phenotype comprising congenital generali