Conflagration
发表于 2025-3-25 03:56:07
Akrodermatitis atrophicans chronica (,),ene oxides as electrophilic reactive metabolites. Much less was known until very recently about the metabolic oxidative activation of thiophene derivatives (Rance, 1989). A first report on the metabolism of thiophene itself in rats has shown the formation of thiophene-derived mercapturates in urine
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发表于 2025-3-25 09:27:19
Pathologische Anatomie der Tuberkulose,ymes are also the major ones involved in the oxidation of potential toxicants and carcinogens such as those encountered among pollutants, solvents, and pesticides, as well as many natural products. A proper understanding of the basic mechanisms by which the P450 enzymes oxidize such compounds is imp
adroit
发表于 2025-3-25 12:15:55
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Oafishness
发表于 2025-3-25 18:48:29
,Acardiacus, Acardius, herzlose Mißgeburt,iety of physiological and pathophysiological processes . Despite of its function in the control of blood pressure, platelet aggregation and neurotransmission, which are mediated via the activation of the soluble guanylate cyclase, ·NO has been shown to be a potent modulator of the cytotoxic acti
变量
发表于 2025-3-25 23:15:57
Muskelrelaxation bei alten Menschen,r, and the rearrangement product chloroacetaldehyde give rise to the etheno (ε) DNA adducts. This type of modified DNA bases appears to be the molecular basis for the genotoxic effects of the parent compound, and site specific mutagenesis studies (Basu et al., 1993) show its mutagenicity. There is e
温室
发表于 2025-3-26 02:12:58
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使苦恼
发表于 2025-3-26 06:06:08
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Mnemonics
发表于 2025-3-26 09:58:48
E. Lüllwitz,U. Dybus,S. Piepenbrockryonic development of all multicellular organisms and also in the adult when various cytokines and hormones exert highly inducer-specific influences on genes. The naturally occurring regulating agents interact with specific receptors: e.g., the retinoids, vitamin D3, thyroid hormones and the steroid
Laconic
发表于 2025-3-26 15:11:56
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conscribe
发表于 2025-3-26 19:52:37
Muskelrelaxanzien und Leberinsuffizienz,DNA binding activity of Fos and Jun is regulated . by a posttranslational mechanism involving reduction-oxidation. Redox regulation is mediated through a conserved cysteine residue located in the DNA binding domain of both proteins. Oxidation or chemical modification of the cysteine has an inhibitor