FISC 发表于 2025-3-30 08:27:39
https://doi.org/10.1007/978-1-84800-054-4They play a critical role in cholesterol disposal and the absorption of fat and fat-soluble vitamins. The concentration of intracellular bile acid is tightly regulated by modulating expression of bile acid transporters via nuclear receptors. This article provides a comprehensive overview of the char火海 发表于 2025-3-30 14:24:26
http://reply.papertrans.cn/19/1863/186221/186221_52.png遗传 发表于 2025-3-30 19:17:41
https://doi.org/10.1007/978-1-84800-054-4 the liver, have been identified as signaling molecules that act as ligands for nuclear receptors, farnesoid X receptor (FXR), pregnane X receptor (PXR), and vitamin D receptor (VDR). FXR is activated by chenodeoxycholic acid, deoxycholic acid, lithocholic acid, cholic acid, and their taurine and glmajestic 发表于 2025-3-31 00:43:05
http://reply.papertrans.cn/19/1863/186221/186221_54.pngtriptans 发表于 2025-3-31 03:19:18
http://reply.papertrans.cn/19/1863/186221/186221_55.pngBasilar-Artery 发表于 2025-3-31 08:18:09
Combinatorics of Colored Dyadic Intervals,long with a global consensus, is a chronic cholestatic liver disease, potentially resulting in liver failure. While the aspect of autoimmune disease of PBC was formally emphasized and immune reactions with lymphocytes against autoantigens were extensively investigated, the interactions between bileIndict 发表于 2025-3-31 10:06:13
Projections, Isomorphisms and Interpolation,nd a poor prognosis. PSC likely occurs in genetically susceptible individuals, perhaps after exposure to environmental triggers including toxic bile acids, bacterial infections, and intestinal pathogens. These could initiate a series of events that involve complex interactions between the innate andRankle 发表于 2025-3-31 16:47:50
Combinatorics of Colored Dyadic Intervals,re, these diseases account for some patients with cholestasis of unknown etiology. Seven known defects of bile acid synthesis occur in children. These defects may be categorized as deficiencies in activity of enzymes catalyzing reactions affecting the steroid nucleus or the side chain. Defects in re