BURSA 发表于 2025-3-25 05:56:22
BLyS/BR3 Receptor Signaling in the Biology and Pathophysiology of Aggressive B-Cell Lymphomas,conservative 发表于 2025-3-25 08:49:51
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Gregg D. Cappon,Donald G. Stump that BR3 and BCR are functionally linked and that Bruton’s cytoplasmic tyrosine kinase (Btk)/NF-κB signaling plays an essential role in this process. Therefore, the primary objective of this article is to discuss BR3-signaling pathways, and the cooperation with BCR signals, that regulate B-cell sur意外 发表于 2025-3-25 17:21:11
https://doi.org/10.1007/978-981-99-1659-7 basis for the unique dependence of B cells on survival signals delivered by BAFF. In the absence of BAFF, B-cell survival is constitutively suppressed through the cooperative actions of the TRAF2 and TRAF3 signal adapters. BAFF circumvents this activity by triggering the recruitment of TRAF3 to BAF确定无疑 发表于 2025-3-25 20:08:14
https://doi.org/10.1007/978-981-99-1659-7ity self-reactive B cells. Interestingly, the expression of the toll-like receptor (TLR) signalling element MyD88 is required for disease, and a reciprocal stimulatory loop exists between TACI and TLR signalling. As TACI is an essential receptor driving T-cell-independent B-cell responses, it raises注射器 发表于 2025-3-26 03:43:14
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Handbook of New Institutional Economicsn the growth and survival of B cells from patients with non-Hodgkin lymphoma, chronic lymphocytic leukemia, multiple myeloma, and Hodgkin lymphoma. Additionally, we will discuss the clinical relevance and regulatory control of BLyS expression in B-cell malignancies.Herd-Immunity 发表于 2025-3-26 09:13:14
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BAFF Receptor Regulation of Peripheral B-Lymphocyte Survival and Development, continually provided by either tonic or antigen-mediated BCR signals and other trophic factors. B-cell-activating factor (BAFF) has emerged as a key growth factor for B lymphocytes. Through its interaction with a TNF-R family member, BAFF-R or BR3, BAFF promotes survival of both immature and maturefiscal 发表于 2025-3-26 16:48:51
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