不怕任性
发表于 2025-3-25 06:29:21
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Inflammation
发表于 2025-3-25 10:39:03
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EWER
发表于 2025-3-25 12:20:06
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换话题
发表于 2025-3-25 17:08:32
yndrome. Designed to provide a detailed and comprehensive visual exposition of all aspects of cardiac function and dysfunction, this atlas contains several hundred images, each accompanied by detailed captions, carefully selected by expert authors, and reviewed by the editor.978-1-4757-4558-0
cacophony
发表于 2025-3-25 21:32:09
Molecular and Cellular Events in Myocardial Hypertrophy and Failure,ers, and peptide growth factors. Genetic manipulations in small animal models are refining our understanding of the stimuli and molecular events that lead to progression of heart failure. Although much remains to be learned about how these stimuli interact with signaling pathways to regulate the rem
支柱
发表于 2025-3-26 01:39:41
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faultfinder
发表于 2025-3-26 07:52:13
Inhibition of the Renin-Angiotensin-Aldosterone System,ent arteriole . Renin system components have been identified in the myocardium and vasculature, where they adversely affect fibrosis and remodeling, as well as cellular dysfunction. These findings suggest that the renin-angiotensinaldosterone system has effects on cardiac function beyond altering
Extricate
发表于 2025-3-26 10:32:35
https://doi.org/10.1007/978-3-642-67575-1ers, and peptide growth factors. Genetic manipulations in small animal models are refining our understanding of the stimuli and molecular events that lead to progression of heart failure. Although much remains to be learned about how these stimuli interact with signaling pathways to regulate the rem
Mortar
发表于 2025-3-26 14:41:03
S. M. Larson,J. S. Rasey,D. R. Allenhamber volume > mass) or a concentric (chamber mass > volume) pattern of ventricular growth. Although remodeling in response to a pathologic condition can in one sense be considered adaptive because it permits the restoration of pump function in the face of an imposed hyperfunctional condition, the
Detoxification
发表于 2025-3-26 18:58:32
Frontiers in Numerical Analysisent arteriole . Renin system components have been identified in the myocardium and vasculature, where they adversely affect fibrosis and remodeling, as well as cellular dysfunction. These findings suggest that the renin-angiotensinaldosterone system has effects on cardiac function beyond altering