ADORN
发表于 2025-3-25 06:14:40
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遗传学
发表于 2025-3-25 09:50:39
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斗志
发表于 2025-3-25 13:50:06
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Insensate
发表于 2025-3-25 19:24:30
James L. Flanaganuss different diseases associated with protein misfolding anAmyloid-forming proteins are implicated in over 30 human diseases. The proteins involved in each disease have unrelated sequences and dissimilar native structures, but they all undergo conformational alterations to form fibrillar polymers.
Climate
发表于 2025-3-25 20:02:17
James L. Flanagane structures, but they all undergo conformational alterations to form fibrillar polymers. The fibrillar assemblies accumulate progressively into disease-specific lesions in vivo. Substantial evidence suggests these lesions are the end state of aberrant protein folding whereas the actual disease-caus
Accommodation
发表于 2025-3-26 02:30:11
James L. Flanagane structures, but they all undergo conformational alterations to form fibrillar polymers. The fibrillar assemblies accumulate progressively into disease-specific lesions in vivo. Substantial evidence suggests these lesions are the end state of aberrant protein folding whereas the actual disease-caus
有权威
发表于 2025-3-26 04:43:42
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PSA-velocity
发表于 2025-3-26 10:34:41
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OUTRE
发表于 2025-3-26 12:40:56
James L. Flanagane structures, but they all undergo conformational alterations to form fibrillar polymers. The fibrillar assemblies accumulate progressively into disease-specific lesions in vivo. Substantial evidence suggests these lesions are the end state of aberrant protein folding whereas the actual disease-caus
悲痛
发表于 2025-3-26 19:10:10
James L. Flanaganuss different diseases associated with protein misfolding anAmyloid-forming proteins are implicated in over 30 human diseases. The proteins involved in each disease have unrelated sequences and dissimilar native structures, but they all undergo conformational alterations to form fibrillar polymers.