–LOUS 发表于 2025-3-26 21:14:29
Nicola Thomasls. In these niches, growth factors and extracellular matrix (ECM) molecules determine the fate of neural stem and progenitor cells (NSPC). However, the precise compounds and the mechanisms that regulate growth factors and other signaling molecules in the niches are unknown. Based on the evidence thFIS 发表于 2025-3-27 04:15:40
ls. In these niches, growth factors and extracellular matrix (ECM) molecules determine the fate of neural stem and progenitor cells (NSPC). However, the precise compounds and the mechanisms that regulate growth factors and other signaling molecules in the niches are unknown. Based on the evidence thHemiparesis 发表于 2025-3-27 06:55:22
http://reply.papertrans.cn/88/8732/873199/873199_33.pngHamper 发表于 2025-3-27 11:46:15
2578-9694 odological exchange between the Anglophone and German traditions of landscape, space and place oriented poetic criticism, to the benefit of both..978-3-030-07963-5978-3-319-90212-8Series ISSN 2578-9694 Series E-ISSN 2634-5188Accommodation 发表于 2025-3-27 14:27:46
Nicola Thomashese factors. Evidence of exogenously transplanted cells functionally integrating into the host brain, replacing cells, and having a behavioural benefit are discussed, along with the ability of some cell sources to stimulate endogenous neuroprotective and restorative events. Alongside exogenous cell山顶可休息 发表于 2025-3-27 17:47:13
Nicola Thomasavioral phenotype that was present in the original 3xTg mouse maintained on a hybrid background. Despite this, the backcrossed 3xTg mice expressed prominent intraneuronal amyloid beta (Aβ) levels in the cortex and amygdala, with lower levels in the CA1 area of the hippocampus. In the combined cohort粘连 发表于 2025-3-27 23:52:52
http://reply.papertrans.cn/88/8732/873199/873199_37.png拒绝 发表于 2025-3-28 05:08:10
http://reply.papertrans.cn/88/8732/873199/873199_38.png谦虚的人 发表于 2025-3-28 07:41:56
Nicola ThomasGF2 at the NSPC surface and likely direct its signaling via tyrosine kinase receptors. Our preliminary results indicate that FGF2 binding to fractone-HSPG is essential for activating FGF2 at the NSPC surface. Moreover, we have found fractones express diverse HSPG at the surface of proliferating NSPC增强 发表于 2025-3-28 14:26:06
GF2 at the NSPC surface and likely direct its signaling via tyrosine kinase receptors. Our preliminary results indicate that FGF2 binding to fractone-HSPG is essential for activating FGF2 at the NSPC surface. Moreover, we have found fractones express diverse HSPG at the surface of proliferating NSPC