持久
发表于 2025-3-23 13:10:19
gainst those from a reasonably standard Genetic Programming (GP) setup. Specifically, the chapter compares a standard GE method to constant creation termed . with what this chapter calls . to constant creation. Constant creation in GE is an important issue due to the disruptive nature of ., which ca
intertwine
发表于 2025-3-23 14:12:21
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mortgage
发表于 2025-3-23 18:49:57
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四海为家的人
发表于 2025-3-24 00:28:50
Octavian Iordache-.-acetyllactosamine (.). Furthermore, the mutant lymphoma cell lines BW5147-PHA.2.1 (.), and KBL-1 (.) are GnT-V-deficient and severely depleted of poly-.- acetyllactosamine on .-glycans, but not the .-glycans. These somatic cell mutants were selected for resistance to the toxic effects of leukoagg
并入
发表于 2025-3-24 03:37:31
Octavian Iordachen gene encoding GnT-I is termed MGAT1 and resides on chromosome 5q35 (.; .), and the mouse gene, Mgat1, is on chromosome 11 (.). Two transcripts of ~2.9kb and ~3.3 kb are observed in most mammalian tissues, with the shorter transcript predominating in liver, and the longer transcript in brain (.; .;
Canary
发表于 2025-3-24 09:39:45
Octavian Iordachegalactosyltransferase (β3GalT) genes have been described that direct the expression of enzymes linking Galβ1,3 to GlcNAc (.; .; .; .; .). A comparison between β3GalT proteins unraveled several conserved domains not found in other galactosyltransferases. Surprisingly, a β3-.-acetylglucosaminyltransfe
喊叫
发表于 2025-3-24 13:01:22
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ingenue
发表于 2025-3-24 18:06:32
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Triglyceride
发表于 2025-3-24 20:45:37
Octavian Iordachee structure of Glcβ1,3Fucα1-Ser/Thr synthesized by POFUT2 for the first Fuc and β3Glc-T for the followed Glc, and further elongation has not been identified. Interestingly, .-fucosylation on TSRs occurs in ER, as opposed to that on the EGF repeat, which occurs in ER and the Golgi apparatus. Biologic
Blood-Clot
发表于 2025-3-25 02:57:45
Octavian Iordached the knockout animals of the GlcT-1 gene proved essential roles in embryo development (Yamashita et al. 1999; Kohyama-Koganeya et al. 2004). However, little is known about why the knockout mouse dies at embryonic day 8 and how GlcT-1 activity is regulated.