根深蒂固 发表于 2025-3-21 17:11:14
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Vasiliki Bitsouni,Nikolaos Gialelis,Ioannis G. Stratisansmembrane anchor) against each other, resulting in membrane fusion. In class II fusion proteins, the fold-back is triggered by the reduced pH of an endosome, and is accompanied by the assembly of fusion protein monomers into trimers. The fold-back occurs by domain rearrangement rather than by an e附录 发表于 2025-3-22 07:22:04
Sergey G. Bobkov,Maria A. Danshina,Vladimir V. Ulyanovfrom Class I or Class II fusion proteins of other viruses. Rhabdoviruses are also notable in that their fusion protein exists in a reversible pH-dependent equilibrium, which prevents irreversible preactivation during assembly, and may prove to be relevant in the mediation of cell-to-cell fusion - anObloquy 发表于 2025-3-22 12:08:36
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Elena Karachanskayaodel of brain injury, the pig. Mechanistic explanations for age- and sex-dependent differences of TBI will focus on the spasmogen endothelin-1, glutamate, and the signaling system mitogen-activated protein kinase. Treatment strategies used for improvement of cerebral hemodynamics after TBI will conshazard 发表于 2025-3-22 19:49:29
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Oleg Kudryavtsevt preconditioning paradigms have been shown to lower TBI-induced damage. Although not all of the mechanisms of preconditioning are confirmed in models of TBI, basic mechanisms of preconditioning applies here as ischemia is a major part of TBI. Ischemic preconditioning, in part, confers protection by烤架 发表于 2025-3-23 04:24:59
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Alexander Melnikov,Hongxi Want preconditioning paradigms have been shown to lower TBI-induced damage. Although not all of the mechanisms of preconditioning are confirmed in models of TBI, basic mechanisms of preconditioning applies here as ischemia is a major part of TBI. Ischemic preconditioning, in part, confers protection by