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Satoshi Kashii,Yoshihito Honda,Akinori Akaikes, including kidney cells. These mechanisms include intracellular sensors for oxidative stress and hypoxia. This means that novel approaches targeting these sensors may offer clinical benefits in kidney disease978-1-4939-5805-4978-1-60761-857-7Series ISSN 2197-7224 Series E-ISSN 2197-7232
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s, including kidney cells. These mechanisms include intracellular sensors for oxidative stress and hypoxia. This means that novel approaches targeting these sensors may offer clinical benefits in kidney disease978-1-4939-5805-4978-1-60761-857-7Series ISSN 2197-7224 Series E-ISSN 2197-7232
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sed Kawasaki disease. An excessive in vivo production of reactive oxygen species results in oxidative stress load, which triggers a never-ending vicious spiral of inflammatory reactions and reactive oxygen metabolites. This forms the basis for acute stage Kawasaki disease. Kawasaki disease involves
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