低位的人或事 发表于 2025-3-28 18:18:57

Microcirculation, NAD/NADH Fluorescence, Extracellular Potassium and Glucose Metabolism Changes in FThe cerebral tissue is very sensitive to interruption of oxygen and substrate supply. In the present study we examined the changes of cerebral electrical activity (ECoG), cerebrocortical blood flow, extracellular potassium concentration (.), NAD/NADH redox state and glucose metabolic rate (CMRg1) in a focal cerebral ischemia model in the cat.

不利 发表于 2025-3-28 20:48:50

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Perigee 发表于 2025-3-29 00:21:47

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特别容易碎 发表于 2025-3-29 04:55:34

https://doi.org/10.1007/978-1-4684-5562-5brain; cell; cell death; cortex; oxygen; paper; physiology; receptor; tissue

Minikin 发表于 2025-3-29 07:57:57

Pathology of Ischaemic Brain Damage — Implications for Therapyter a stroke. The second important step in the treatment of acute stroke is inhibition of blood platelet aggregation to arrest the thrombotic process and avoid complete occlusion of the artery, or in the case of an embolism to limit the extent of the infarct.

幼稚 发表于 2025-3-29 15:29:30

Cerebral Hypoxia during Repetitive Seizurestatus epilepticus (e.g., pulmonary edema) also can profoundly decrease cerebral oxygenation. An important remaining question is whether the cerebral hypoxia accompanying later seizures contributes to the neuronal damage following prolonged status epilepticus.

Control-Group 发表于 2025-3-29 16:14:21

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ellagic-acid 发表于 2025-3-29 23:31:37

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Cantankerous 发表于 2025-3-30 01:37:26

Cerebral Blood flow and Its Responsiveness to CO2 after Traumatic and Ischemic Brain Injuries tissue electric impedance (cf refs 1,2). Measurements by REG roughly matched those by H. clearance. Measurements were made while the animal was breathing room air and also during 7% CO. inhalation, which raised PaCO. by 6–8 mmHg Responsiveness to CO. was quantified as the ratio of rCBF with and without elevated PaCO..

偏离 发表于 2025-3-30 05:30:20

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查看完整版本: Titlebook: Mechanisms of Cerebral Hypoxia and Stroke; George Somjen Book 1988 Plenum Press, New York 1988 brain.cell.cell death.cortex.oxygen.paper.p