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https://doi.org/10.1007/978-1-59259-703-1Antigen; apoptosis; autoimmunity; bacteria; cytokine; genetics; inflammation; lymphocytes; macrophages; patho商谈 发表于 2025-3-27 05:49:30
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Genetic Insights into Murine Lupus,tinguish” self antigens from nonself is the end result of an elaborate network of interrelated pathways that function to “tolerize” antiself B and T cells. Indeed, potentially autoreactive lymphocytes have been documented to be weeded out through a systematic array of B- and T-cell tolerance checkpoints (reviewed in refs. . and .).睨视 发表于 2025-3-27 17:32:45
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The Vascular Endothelium,es remain paramount in the pathogenesis of inflammation, the endothelial cell, once thought to be a passive bystander, is now recognized as one of the central, active participants in the inflammatory process. The endothelium dictates the site of inflammation and the types of cells involved in the indelta-waves 发表于 2025-3-28 11:44:25
Nitric Oxide in Systemic Lupus Erythematosus, erythematosus (SLE) .. Although the inhibition of NO synthesis improves outcome in the MRL/. mice, the role of NO in the pathogenesis of SLE is as yet undetermined. The biology of NO, produced by constitutive or induced enzymes in virtually all tissues, is likely to exert complex actions on both im