忧伤
发表于 2025-3-25 03:34:37
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无节奏
发表于 2025-3-25 08:28:50
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很像弓]
发表于 2025-3-25 14:17:37
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Apoptosis
发表于 2025-3-25 16:26:22
Phenotypic Heterogeneity and the Origins of Lymphoid Malignancy in Man: Biological and Clinical Impukemia phenotypes (excluding chromosome changes) appear to be minimally deviated replicas of normal ones. Uncoupling of proliferation from differentiation in acute leukemia results in the stabilization of progenitor cell phenotypes that would ordinarily be very transient. The phenotypic abnormality
SOW
发表于 2025-3-25 23:17:09
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牛马之尿
发表于 2025-3-26 03:24:34
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最高点
发表于 2025-3-26 07:39:22
Multiple Mechanisms of Oncogene Activation in Viral and Nonviral Neoplasia,equences, gene amplification, translocation, and point mutations within regulatory or coding sequences. The mechanisms by which a specific proto-oncogene can most readily be activated probably reflects unique structural and biological characteristics of the gene involved. The c-. gene can be activat
Dedication
发表于 2025-3-26 11:48:05
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影响带来
发表于 2025-3-26 12:54:02
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grandiose
发表于 2025-3-26 20:28:43
Tracing Development and Cell Lineages in Human Hemopoietic Neoplasia,tic neoplasms. The data indicate that chronic myelocytic leukemia (CML) is a clonal disease that involves a stem cell pluripotent for B lymphoid as well as myeloid cells. Although T cells may also arise from this stem cell, this question has not been definitively resolved. Elements that make up the