行为 发表于 2025-3-23 10:21:55
http://reply.papertrans.cn/51/5003/500294/500294_11.png巨硕 发表于 2025-3-23 14:18:36
http://reply.papertrans.cn/51/5003/500294/500294_12.pngFECK 发表于 2025-3-23 20:20:12
http://reply.papertrans.cn/51/5003/500294/500294_13.pngMendacious 发表于 2025-3-24 01:11:49
lus complement and transplanted into anemic mice produces sufficient numbers of macroscopic colonies on the surface of spleens, but these colonies are predominantly committed granulocyte precursor elements with no potential to produce stem cells (17,18).airborne 发表于 2025-3-24 05:37:32
Stefanie Averbecklus complement and transplanted into anemic mice produces sufficient numbers of macroscopic colonies on the surface of spleens, but these colonies are predominantly committed granulocyte precursor elements with no potential to produce stem cells (17,18).CUB 发表于 2025-3-24 08:58:22
http://reply.papertrans.cn/51/5003/500294/500294_16.pngConfidential 发表于 2025-3-24 12:20:47
Ernst Manheimlus complement and transplanted into anemic mice produces sufficient numbers of macroscopic colonies on the surface of spleens, but these colonies are predominantly committed granulocyte precursor elements with no potential to produce stem cells (17,18).CYN 发表于 2025-3-24 18:34:46
Ernst Manheim it is a relatively poor stimulator of CFU-c (6, 12). Third, 4-methyl-hista- mine, which has been shown by Byron (1) to cause murine CFU-s to start DNA synthesis, has been shown to have a similar effect on the human ΔC progenitors, but to have no detectable effect on the cell cycle phase of human CFDetoxification 发表于 2025-3-24 19:12:28
Ernest Manheim complex of at least two biologic entities (6,17): the spleen focus-forming virus (SFFV), which is thought to be responsible for the increase in erythropoiesis, and a murine leukemia virus (MuLV), which can be isolated free of SFFV and which produces a leukemia not accompanied by an increase in eryt树木心 发表于 2025-3-25 01:29:33
Carsten Klingemann,Michael Neumann,Karl-Siegbert Rehberg,Ilja Srubar,Erhard Stölting it is a relatively poor stimulator of CFU-c (6, 12). Third, 4-methyl-hista- mine, which has been shown by Byron (1) to cause murine CFU-s to start DNA synthesis, has been shown to have a similar effect on the human ΔC progenitors, but to have no detectable effect on the cell cycle phase of human CF